Kelainan jantung
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Transcript of Kelainan jantung
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04/08/13 1
PENYAKIT-PENYAKIT PADA JANTUNG
1. Coronary heart disease ± 80%
2. Hipertensive heart disease ± 9%
3. Rheumatic heart disease ± 2-3%
4. Congenital heart disease ± 2%
5. Bacterial endocarditis ± 1-2%
6. Sifilitik heart disease ± 1%
7. Cor pulmonale ± 1%
8. Penyakit jantung lainnya ± 5%
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1. PENYAKIT JANTUNG KORONER
Gangguan pada myocardium biasanya disebabkan coronary insuffisience
- arteriosclerosis 99%
- rheumatica
- sifilis
- arteritis
- polyarteritis
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Varian penyakit jantung koroner :
1. Arteriosclerotic heart disease
2. Angina pectoris
3. Myocardial infarction
Dipengaruhi oleh :
1. Aliran darah pada a. coronary
2. Sensitivitas myocardium terhadap ischaemia
3. Konsentrasi O2 dalam darah
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04/08/13 6
Keadaan yang dihubungkan dengan Penyakit jantung Koroner:
1. Hypertensi
2. Obesitas
3. Hypercholesterolemia
4. Merokok
5. DM
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1. insufisiensi coronary disebabkan oleh :
a. Aortitis luetica
b. Jaringan granulasi pada a. proximal
c. A. coronaria aneurysm
d. Buerger disease
e. Polyarteritis nodosa/ Rheumatica
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2. Aktifitas myocardial dipengaruhi oleh :
a. Hyperthyroid
b. Kehamilan
c. Hypermetabolic, febris
d. Exercise
3. Konsentrasi O2 :
a. Anemia berat
b. kelainan pada eritrosit, polycythemia
c. Cyanosis
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Faktor Predisposisi:
1. Lipoprotein serum ↑, soft drink, obesitas, alkohol
2. Tekanan darah meningkat
3. Peningkatan kadar gula darah
4. Stress
5. Kurang olah raga
6. Merokok
7. Uric acid serum ↑
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ARTERIOSCLEROTIC HEART DISEASE
Arteriosclerosis pada a. coronary & fibrosis diffuse myocardial & sering disertai dengan fibrosis pada katub jtg
Morfologi :
Arteriosclerotic ischaemia otot myocardial fibrosis yang tampak sebagai plaque abu-abu yang diffuse
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atrofi pada serabut otot myocardium dan dijumpai lipochrom = Brown atrophy sehingga jantung dapat menjadi :
mengecil normal membengkak (pada Decompensatio
Cordis )
Abnormalitas pada katub : Fibrosis katub mitral Fibrosis chordae tendinea kalsifikasi
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MANIFESTASI KLINIKasymptomatisangina pectorismitral / aortic murmurkerusakan myocardium yg terlihat pada
EKGkongesti jantungaritmia and myocardium infark
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hipoksia pada myocardium dapat terjadi pada peny. : coronary arteriosclerosis myocardium luetica polyarthritis nodosa insufisiensi katup aorta anemia
Hypoxia disebabkan oleh : Occlusion pada arteri kecil Spasme arteri Pada saat beraktivitas hypoxia paroxysmal
myocardium
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ANGINA PECTORIS
Merupakan simptom klinik yang ditandai dengan serangan nyeri paroksismal yang temporer pada substernal ataupun precordial dan biasanya terjadi setelah exercise dan hilang bila istirahat.
Tidak tampak kerusakan myocardiumEKG normal
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MYOCARDIUM INFARCT ( MCI )
insufisiensi Coronary disebabkan oleh :
coronary arteriosclerotic 99 % thrombosis and embolismPenyakit pada pembuluh darahPenyempitan ostium padasyphilis Arteriosclerosis dan hypertensi
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MYOCARD INFARK (MCI)MYOCARD INFARK (MCI)
Predileksi : right a. coronary 40 % left anterior a. coronary 40 % Ventrikel kiri
Morfologi : Restriksi pada central myocardium Myocardium : epicardium + endocardium
menjadi menebal (Ø 3 – 4 cm). Zahn Infarction, lesi kecil subendocardial
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Myocardium Infarct Progressivity :Myocardium Infarct Progressivity :< 12 hours : vague or pale 18 - 24 hours : clearly anemic, brown-grey,
stable muscle consistency. 2 – 4 days : well defined necrotic tissue
border surround by hyperaemic area, soft, yellow in colour, due to fatty changes
4 – 10 days : progressive fatty degeneration, central necrosis, soft ,haemorrhage (greyish yellow), well -defined border
6 weeks : fibrosis
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Mikroskopis :
Iskemik pembulu darah → koagulasi nekrosis pada sel miokardium
interstitial edema haemorrhage / haemosiderin pigmen Eksudasi netrofil fibrosis
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KOMPLIKASI :
Pericarditis fibrinous / haemorrhagica Mural Thrombosis → embolism Rupture infarction → heart tamponade Fibrosis dan aneurysm
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Lab :
Necrosis coagulativa enzyme dehidrogenase ↑glutamic oxaloacetic transaminase ↑
12 – 24 jam• SGOT ↑• Leucocytocis ↑• BSR ↑• LDH ↑• C Reactive Protein ↑
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MANIFESTASI KLINIK:Nyeri yang tiba-tiba dan dalam pada
substernal dan precordial.Nyeri menyebar pada punggung kiri ,
lengan sampai ke jari-jari dan pada daerah dagu.
Rasa seperti tertekan, berkeringat , mual, muntah
lemasAliran darah ↓ sampai shockDyspnoe
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CyanotisAritmia jantungECG : - Q abnormal
- ST elevation - T invertedKematian : male >> 2 x female
Insiden : Laki-laki : 33- 35 tahun Wanita : setelah menopause
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HYPERTENSIVE HEART DISEASE
Ditandai dengan hypertrofi ventrikel kiri, oleh karena continous systemic hypertensive.
Insiden : Female >> 2x male sering terjadi pada usia pertengahan/tua Genetically inherited
Etiologi :Hypertensi dapat terjadi apabila resistensi arterial peripheral ↑
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Peningkatan resistensi periferal disebabkan oleh : vasokontriksi pembuluh darah arteriole, arteri
kecil Penyakit pada pembuluh darah yang diffuse
Hypertensi → hypertrofi myocardium → coronary arteriosclerotic resistensi perifer ↑ jantung akan mengalami
compensated cardiac output → hypertrofi myocardium → ventrikel kiri membesar → dilatasi jantung
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Hypertrofi pada otot jantung disebabkan oleh :Aktivitas >>→ anoxia kelemahan myocard hipertensi → coronary arteriosclerosis →
myocardium anoxia.hipertensi → kerusakan pada pembuluh darah
renal → Renin ↑ → NaCl + H2O retensi
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Morfologi :
Dinding ventrikel kiri >> ( 2,5 cm )Berat jantung >>Tidak disertai kelainan jantung lainnyamikroskopik : otot jantung normal Penebalan dinding arteri
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MANIFESTASI KLINIK :
Stadium kompensasi : asimptomatisHypertensive symptom :
sakit kepala, palpitasi, retinopathyDecompensatio jantung kiri:
dyspnoe, batuk, batuk darahSimptom Coronary arteriosclerose
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RHEUMATIC HEART DISEASERHEUMATIC HEART DISEASE
Demam rematik merupakan penyakit peradangan sistemik non supurative .
Dihubungkan dengan infeksi Streptococcus beta haemolitycus group A dan reaksi imunologi pada saat demam dan remisi yang berkepanjangan .
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Demam rheumatic merupakan penyakit kolagen , dapat terjadi padabagian tubuh :
persendian jantung kulit serous Pembuluh darah paru
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Insiden : usia 5 – 15 tahun 90 % Sosial ekonomi rendah Daerah padat penduduk, sanitasi buruk Nutrisi kurang baik
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Perubahan pada peradangan rheumatic
mucoid degenerasifibrinoid nekrosishyaline collagen degenerasi
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Etiologi:
Demam rheumatik terjadi setelah1 - 4 minggu terinfeksi oleh streptococcus
( Pharyngitis, Tonsilitis, Scarlatina ) Reaksi antigen - antibody
menyebabkan allergi fokal nekrosis. Ditandai oleh kadar ASTO ↑
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Morfology:Kelainan spesifik dan patognomonik:
Aschoff body → degenerasi focus fibrinoid yang dikelilingi oleh infiltasi sel radang
Focus peradangan : - jantung, - sendi synovial
- fascia tendonVegetation nodule dapat dijumpai pada
kulit , subcutis Ø 1 -4 cm subcutaneous nodule
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Katub mitral, aortic : fibrotic vegetation, kalsifikasi
Katub tricuspidale : stenosis & insuffisiensi
Chordae tendinea : memendek dan menebal
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Clinical ManifestationClinical Manifestation :
Major Criteria of Jones:
1. Polyarthritis migrans 85 %
2. Carditis 65 %
3. Chorea Sydenham
4. Subcutaneous nodule
5. Erythema marginatum
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Minor Criteria of Jones
1. Leucocytosis
2. BSR ↑
3. ASTO ↑
4. Demam
5. Arthralgia
6. Prolonged PR interval
7. Erythema
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Penyebab kematian :
1. Decompensatio cordis
2. Brain/ renal thromboembolism
3. Bacterial endocarditis
4. Mitral stenosis
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COR PULMONALE
Hypertrofi ventrikel kanan , yang disebabkan oleh penyakit – penyakit pulmonal
Etiologi :
1. Acute massive pulmonary embolism, sehingga menimbulkan dilatasi pada ventrikel kanan
2. Chronic 1. Pulmonary disease
2. Chronic pulmonary disease
3. Thorax abnormalities
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1. Penyakit pada paru
- Diffuse trombosis pada arteri besar/kecil
- Embolism
- Diffuse vasculitis
- Fibrosis : - sarcoidosis
- radiation
- asbestosis
- berryliosis
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2. Penyakit paru kronik
- Emphysema
- Chronic bronchitis
- Pulmonary fibrosis oleh karena TBC
- Sarcoidosis
- Severe pneumonia
- Pulmonary resection
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– penebalan pleura bilateral - Neuro abnormalities :
PoliomyelitisMyasthenia gravisMuscle distrophyKyphoscoliosis
Manifestasi Klinik:- dyspnoe - dilatasi vena- oedema - ascites- hydrothorax - hepatosplenomegaly
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CONGENITAL HEART DISEASE
Etiology :
- Tidak diketahui
- Genetics
- Infeksi rubella, lues, toxoplasma
- Obat-obatan teratogenic thalidomide, cortison, busulfan
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1. Roger’s disease = Ventricle Septal DefectManifest in IV-VII weeksMacros : - defect, mmcm
- ventrikel kanan >> - penebalan endocardium
defect parallel
Klinis : - hard systole murmur = machinary murmur
- pulmonary hypertension - tardive cyanosis04/08/13 44
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Penyebab kematian : - right DC - endocarditis
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2. Atrium Septal Defect
Setelah minggu ke IV
Defect pada foramen ovale
Klinis : - cyanotic right sided overload
- hypertrophy right ventricle
- pulmonary hypertension
- systolic murmur
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3. Lutembacher’s disease - ASD + Stenosis mitral - dilatasi ventrikel kiri dan kanan - hypertrofi ventrikel kanan
4. Tetralogi Fallot- Defect septum interventricle- Dextroposed overriding aorta- Stenosis katub pulmonal- hypertrophy ventrikel kanan
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Manifestasi Klinis : cyanosis sejak lahir clubbing fingers gangguan tumbuh kembang
Prognosis jelek dan dapat menyebabkan kematian , bila :
- Decompensatio Cordis kanan- Endocarditis bacterialis- Abcess pada otak - Respiratory infection
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5. Eisenmenger Complex
Varian dari Tetralogi Fallot tetapi tanpa disertai stenosis pulmonalis
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6. Patent Ductus Arteriosus
- Ductus Botalli menghubungkan a.pulmonale & aorta
- Ductus Botalli mulai tertutup pada usia 1-2 tahun setelah lahir
- Aliran darah dari aorta ke a.pulmonalis, dapat berkurang pada sirkulasi
- hypertrofi ventrikel kanan
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COARCTATIO AORTA
Aortic Stenosis hypertrofi ventrikel kiri proximal dilatasi, darah >> sakit
kepala distal vasokonstriksi extremitas
pucat & dingin
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PERICARDIUM
Cairan ↓↓ pada cavum pericardium
1. HydropericardiumNormal : 30-50cc, serous
< 50cc - D.C, - Penyakit ginjal kronik
- Hypoproteinemia
chronic pericard adhesion
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2. Hemopericardium
Darah >> pericardium, oleh karena : TraumaRupture of muscle infark myocardRupture of aorta Malignant tumorRupture of a. coronary
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PERICARDITIS
Biasanya merupakan reaksi sekunder pd penyebaran infeksi : - hematogen
- lymphogen
- percontuinitatum
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Morfologi :
- dilatasi & irregularity pada vena, ectasion
- penebalan katub
- penebalan dinding yang tidak teratur
- perubahan jaringan elastik oleh karena proses fibrosis
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Classification of pericardium based of etiology : Tuberculosis pericarditis Bacterialis pericarditis Rheumatica pericarditis Uremic pericarditis Virus pericarditis Carcinomatosa pericarditis MCI yang menyebabkan pericarditis Infeksi sekunder : - parasit - fungal Idiopathic
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Klasifikasi berdasarkan peradangan exudate :
1. Serous pericarditis 2. Serofibrinous pericarditis
3. Fibrinous pericarditis
4. Suppurative pericarditis
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Pathogenesis
1. Anomalies jantung MCI
Acute rheumatica
Surgical trauma
2.Pulmonary disease Tuberculosis
Carcinoma
Pneumonia
Empyema
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Komplikasi:
1. Constrictive pericarditis
2. Obliterative, focal/diffuse pericarditis
3. V. cava compression
- ascites
- hepatosplenomegaly
4. DC
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Terima kasih