PATIENT NAME : MRS. KIRAN SHARMA - GiveIndia

Cert. No. MC-2461

PATIENT NAME : MRS. KIRAN SHARMA

PATIENT ID : FH.10727635

ACCESSION NO : 0013UE004728 AGE : 60 Years SEX : Female DATE OF BIRTH : 01/01/1961

DRAWN : 21/05/2021 06:41 RECEIVED : 21/05/2021 07:15 REPORTED : 21/05/2021 08:58

REFERRING DOCTOR : DR. ER Doctor Charges

CLIENT PATIENT ID : UID:10727635

CLIENT NAME : FLT LT RAJAN DHAL CHARITABLE

TRUST IPD

UID:10727635 REQNO-6397811

IPD-EMERGENCY

IPID-63236/21/1301

CLINICAL INFORMATION :

Final Results Biological Reference Interval UnitsTest Report Status

HAEMATOLOGY

COMPLETE BLOOD COUNT, EDTA WHOLE

BLOOD/SMEAR

BLOOD COUNTS

HEMOGLOBIN 12.6 12.0 - 15.0 g/dL

METHOD : PHOTOMETRIC

RED BLOOD CELL COUNT 4.70 3.8 - 4.8 mil/µL

METHOD : ELECTRICAL IMPEDANCE

WHITE BLOOD CELL COUNT 23.26 4.0 - 10.0 thou/µLHigh


PLATELET COUNT 175 150 - 410 thou/µL


Comments

GIANT PLATELET SEEN.

RBC AND PLATELET INDICES

HEMATOCRIT 37.47 36 - 46 %

METHOD : MEASURED

MEAN CORPUSCULAR VOL 79.7 83.0 - 101.0 fLLow

METHOD : MEASURED

MEAN CORPUSCULAR HGB. 26.7 27.0 - 32.0 pgLow

METHOD : CALCULATED PARAMETER

MEAN CORPUSCULAR HEMOGLOBIN

CONCENTRATION

33.5 31.5 - 34.5 g/dL


RED CELL DISTRIBUTION WIDTH 17.5 11.6 - 14.0 %High


MEAN PLATELET VOLUME 12.5 6.8 - 10.9 fLHigh


WBC DIFFERENTIAL COUNT - NLR

SEGMENTED NEUTROPHILS 89 40 - 80 %High

METHOD : VCS TECHNOLOGY/ MICROSCOPY

ABSOLUTE NEUTROPHIL COUNT 20.70 2.0 - 7.0 thou/µLHigh


LYMPHOCYTES 3 20 - 40 %Low


ABSOLUTE LYMPHOCYTE COUNT 0.70 1.0 - 3.0 thou/µLLow


NEUTROPHIL LYMPHOCYTE RATIO (NLR) 30.9

Of 2C/O FORTIS RAJAN DHALL HOSPITAL,SECTOR B,POCKET 1,ARUNA ASAF ALI MARG, VASANT KUNJ

NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITEDC/O FORTIS RAJAN DHALL HOSPITAL,SECTOR B,POCKET 1,ARUNA ASAF ALI MARG, VASANT KUNJ

NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

Cert. No. MC-2461








TRUST IPD

UID:10727635 REQNO-6397811

IPD-EMERGENCY

IPID-63236/21/1301



EOSINOPHILS 1 1 - 6 %


ABSOLUTE EOSINOPHIL COUNT 0.23 0.02 - 0.50 thou/µL


MONOCYTES 3 2 - 10 %


ABSOLUTE MONOCYTE COUNT 0.70 0.2 - 1.0 thou/µL


BASOPHILS 0 < 1 - 2 %

ABSOLUTE BASOPHIL COUNT 0.00 0.02 - 0.10 thou/µLLow


BAND (STAB) CELLS 03 %

MYELOCYTES 01 %High

DIFFERENTIAL COUNT PERFORMED ON: EDTA SMEAR

METHOD : AUTOMATED ANALYZER / MICROSCOPY

DISCLAIMER: THE ABSOLUTE WHITE CELL COUNTS ARE OUTSIDE THE NABL ACCREDITED SCOPE OF THE LABORATORY.

Interpretation(s)

BLOOD COUNTS-

The cell morphology is well preserved for 24hrs. However after 24-48 hrs a progressive increase in MCV and HCT is observed leading to a decrease in MCHC. A direct smear is

recommended for an accurate differential count and for examination of RBC morphology.

RBC AND PLATELET INDICES-



WBC DIFFERENTIAL COUNT - NLR-

The optimal threshold of 3.3 for NLR showed a prognostic possibility of clinical symptoms to change from mild to severe in COVID positive patients. When age = 49.5 years

old and NLR = 3.3, 46.1% COVID-19 patients with mild disease might become severe. By contrast, when age < 49.5 years old and NLR < 3.3, COVID-19 patients tend to

show mild disease.

(Reference to - The diagnostic and predictive role of NLR, d-NLR and PLR in COVID-19 patients ; A.-P. Yang, et al.; International Immunopharmacology 84 (2020) 106504

This ratio element is a calculated parameter and out of NABL scope.

**End Of Report**

Please visit www.srlworld.com for related Test Information for this accession

Dr Nitin Kumar Dumeer, M.D.

Senior pathologist,Lab head

Of 2C/O FORTIS RAJAN DHALL HOSPITAL,SECTOR B,POCKET 1,ARUNA ASAF ALI MARG, VASANT KUNJ

NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

Cert. No. MC-2461





REFERRING DOCTOR :



TRUST IPD

UID:10727635 REQNO-6398686

IPD-ICU(4)

IPID-63236/21/1301



COAGULATION

ACT PARTIAL THROMBO PLASTIN TIME

(APTT), PLASMA

APTT 22.3 22.2 - 33.2 SECONDS

METHOD : CLOT DETECTION BY BALL OSCILATION

Comments

LOW OR HIGH HEMATOCRIT VALUES CAN ALTER PT & APTT VALUE.

KINDLY CORRELATE CLINICALLY.

PROTHROMBIN TIME, PLASMA

PROTHROMBIN TIME (PT) 15.0 11.6 - 14.6 SECONDSHigh

METHOD : CLOT DETECTION BY BALL OSCILATION

INTERNATIONAL NORMALIZED RATIO (INR) 1.18 < 1.4 RATIO

MEAN NORMAL PT 13.1 SECONDS

Interpretation(s)

ACT PARTIAL THROMBO PLASTIN TIME(APTT), PLASMA-

The activated partial thromboplastin time (APTT) reflects the activities of most of the coagulation factors, including factor XII and other ""contact factors"" (prekallikrein [PK]

and high molecular weight kininogen [HMWK]) and factors XI, IX, and VIII in the intrinsic coagulation pathway, as well as coagulation factors in the common coagulation

pathway that include factors X, V, II and fibrinogen (factor I). The APTT also depends on phospholipid (a partial thromboplastin) and ionic calcium, as well as the activator of

the contact factors (eg, silica) present in the reagent, but reflects neither the integrity of the extrinsic coagulantion pathway that includes factor VII and tissue factor, nor the

activity of factor XIII (fibrin stabilizing factor). The APTT is variably sensitive to the presence of specific and nonspecific inhibitors of the intrinsic and common coagulation

pathways, including lupus anticoagulants or antiphospholipid antibodies. It is useful for monitoring unfractionated heparin therapy, for screening for certain coagulation factor

deficiencies, detection of coagulation inhibitors such as lupus anticoagulant, specific factor inhibitors, and nonspecific inhibitors.

APTT “mixing” studies:

Poor or partial correction of the abnormal result by normal plasma may be observed in the presence of coagulation factor inhibitors, anticoagulant drugs such as heparin or

direct thrombin inhibitors. Total correction indicates coagulation factors deficiency.

PROTHROMBIN TIME, PLASMA-

Prothrombin Time measures the integrity of the extrinsic pathway and the adequacy of critical coagulation factors involved in it, namely, Factor VII. This test is therefore,

used for monitoring oral anticoagulation therapy which lowers the levels of multiple vitamin K dependent coagulation factors in blood (Factors II, VII, IX and X) including

Factor VII. The result of PT is expressed as International Normalized Ratio (INR) to neutralize the influence of variable sensitivity of the reagents (thromboplastin) used in the

assay by different laboratories.

Prolonged PT/INR is observed in hereditary or acquired deficiency of the relevant coagulation factors, vitamin K deficiency, liver disease, specific coagulation factor inhibitors

and nonspecific inhibitors of PT (eg, monoclonal immunoglobulins, elevated fibrin degradation products).

The following INR ranges are recommended for achieving optimal anticoagulation in different clinical conditions:

Diagnosis TargetINR

Treatment of venous thrombosis 2.0- 3.0

Treatment of pulmonary embolism 2.0- 3.0

Prevention of systemic embolism 2.0- 3.0

Tissue heart valves 2.0- 3.0

Hypercoagulable states 2.0- 3.0

Atrial fibrillation 2.0- 3.0

Mechanical prosthetic valves (high risk) 2.5- 3.5

Bileaflet mechanical valve in aortic position 2.0- 3.0

Page 1 Of 2C/O FORTIS RAJAN DHALL HOSPITAL,SECTOR B,POCKET 1,ARUNA ASAF ALI MARG, VASANT KUNJ

NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

Cert. No. MC-2461





REFERRING DOCTOR :



TRUST IPD

UID:10727635 REQNO-6398686

IPD-ICU(4)

IPID-63236/21/1301



**End Of Report**





NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

Cert. No. MC-2461








TRUST IPD

UID:10727635 REQNO-6397939

IPD-EMERGENCY

IPID-63236/21/1301



KIDNEY PANEL - 1

SERUM BLOOD UREA NITROGEN

BLOOD UREA NITROGEN 15 8 - 23 mg/dL

METHOD : UREASE -GLDH

CREATININE EGFR- EPI

CREATININE 0.62 0.60 - 1.20 mg/dL

METHOD : MODIFIED JAFFE KINETIC

AGE 60 years

METHOD : MANUAL

GLOMERULAR FILTRATION RATE (FEMALE) 98.01 Refer Interpretation Below mL/min/1.73m2


BUN/CREAT RATIO

BUN/CREAT RATIO 24.19 5.00 - 15.00High


URIC ACID, SERUM

URIC ACID 4.6 2.6 - 6.0 mg/dL

METHOD : URICASE/ PAP

TOTAL PROTEIN, SERUM

TOTAL PROTEIN 5.8 6.4 - 8.2 g/dLLow

METHOD : BIURET

ALBUMIN, SERUM

ALBUMIN 2.2 3.4 - 5.0 g/dLLow

METHOD : BROMOCRESOL PURPLE

GLOBULIN

GLOBULIN 3.6 2.0 - 4.1 g/dL


ELECTROLYTES (NA/K/CL), SERUM

SODIUM 131 136 - 145 mmol/LLow

METHOD : ION SELECTIVE ELECTRODE TECHNOLOGY INDIRECT

POTASSIUM 3.40 3.50 - 5.10 mmol/LLow


CHLORIDE 96 98 - 107 mmol/LLow


URINALYSIS

COLOR PALE YELLOW

METHOD : MANUAL

APPEARANCE CLEAR

METHOD : MANUAL


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

Cert. No. MC-2461








TRUST IPD

UID:10727635 REQNO-6397939

IPD-EMERGENCY

IPID-63236/21/1301



PH 6.0 4.7 - 7.5

METHOD : DIPSTICK

SPECIFIC GRAVITY 1.015 1.003 - 1.035

METHOD : DIPSTICK

GLUCOSE NOT DETECTED NOT DETECTED

METHOD : DIPSTICK / MANUAL

PROTEIN NOT DETECTED NOT DETECTED


KETONES NOT DETECTED NOT DETECTED


BLOOD NOT DETECTED NOT DETECTED

METHOD : DIPSTICK

BILIRUBIN NOT DETECTED NOT DETECTED


UROBILINOGEN NORMAL NORMAL


NITRITE NOT DETECTED NOT DETECTED

METHOD : DIPSTICK

WBC 1-2 0-5 /HPF

METHOD : MICROSCOPIC EXAMINATION

EPITHELIAL CELLS 1-2 0-5 /HPF


RED BLOOD CELLS NOT DETECTED NOT DETECTED /HPF


CASTS NOT DETECTED


CRYSTALS NOT DETECTED


Comments

NOTE :- MICROSCOPIC EXAMINATION OF URINE IS PERFORMED BY CENTRIFUGED URINARY SEDIMENT.

Interpretation(s)

SERUM BLOOD UREA NITROGEN-

Causes of Increased levels

Pre renal

• High protein diet, Increased protein catabolism, GI haemorrhage, Cortisol, Dehydration, CHF Renal

• Renal Failure

Post Renal

• Malignancy, Nephrolithiasis, Prostatism

Causes of decreased levels

• Liver disease

• SIADH.

CREATININE EGFR- EPI-

GFR— Glomerular filtration rate (GFR) is a measure of the function of the kidneys. The GFR is a calculation based on a serum creatinine test. Creatinine is a muscle waste


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

C/O FORTIS RAJAN DHALL HOSPITAL,SECTOR B,POCKET 1,ARUNA ASAF ALI MARG, VASANT KUNJ

NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

Cert. No. MC-2461








TRUST IPD

UID:10727635 REQNO-6397939

IPD-EMERGENCY

IPID-63236/21/1301


Final Results Biological Reference IntervalTest Report Status

product that is filtered from the blood by the kidneys and excreted into urine at a relatively steady rate. When kidney function decreases, less creatinine is excreted and

concentrations increase in the blood. With the creatinine test, a reasonable estimate of the actual GFR can be determined.

A GFR of 60 or higher is in the normal range.

A GFR below 60 may mean kidney disease.

A GFR of 15 or lower may mean kidney failure.

Estimated GFR (eGFR) is the preferred method for identifying people with chronic kidney disease (CKD). In adults, eGFR calculated using the Modification of Diet in Renal

Disease (MDRD) Study equation provides a more clinically useful measure of kidney function than serum creatinine alone.

The CKD-EPI creatinine equation is based on the same four variables as the MDRD Study equation, but uses a 2-slope spline to model the relationship between estimated GFR

and serum creatinine, and a different relationship for age, sex and race. The equation was reported to perform better and with less bias than the MDRD Study equation,

especially in patients with higher GFR. This results in reduced misclassification of CKD.

The CKD-EPI creatinine equation has not been validated in children & will only be reported for patients = 18 years of age. For pediatric and childrens, Schwartz Pediatric

Bedside eGFR (2009) formulae is used. This revised "bedside" pediatric eGFR requires only serum creatinine and height.

URIC ACID, SERUM-


Dietary

• High Protein Intake.

• Prolonged Fasting,

• Rapid weight loss.

Gout

Lesch nyhan syndrome.

Type 2 DM.

Metabolic syndrome.


• Low Zinc Intake

• OCP’s

• Multiple Sclerosis

Nutritional tips to manage increased Uric acid levels

• Drink plenty of fluids

• Limit animal proteins

• High Fibre foods

• Vit C Intake

• Antioxidant rich foods

TOTAL PROTEIN, SERUM-

Serum total protein,also known as total protein, is a biochemical test for measuring the total amount of protein in serum..Protein in the plasma is made up of albumin and

globulin

Higher-than-normal levels may be due to: Chronic inflammation or infection, including HIV and hepatitis B or C, Multiple myeloma, Waldenstrom's disease

Lower-than-normal levels may be due to: Agammaglobulinemia, Bleeding (hemorrhage),Burns,Glomerulonephritis, Liver disease, Malabsorption, Malnutrition, Nephrotic

syndrome,Protein-losing enteropathy etc.

ALBUMIN, SERUM-

Human serum albumin is the most abundant protein in human blood plasma. It is produced in the liver. Albumin constitutes about half of the blood serum protein. Low blood

albumin levels (hypoalbuminemia) can be caused by: Liver disease like cirrhosis of the liver, nephrotic syndrome, protein-losing enteropathy, Burns, hemodilution, increased

vascular permeability or decreased lymphatic clearance,malnutrition and wasting etc.

ELECTROLYTES (NA/K/CL), SERUM-

Sodium levels are Increased in dehydration, cushing's syndrome, aldosteronism & decreased in Addison's disease, hypopituitarism,liver disease. Hypokalemia (low K) is

common in vomiting, diarrhea, alcoholism, folic acid deficiency and primary aldosteronism. Hyperkalemia may be seen in end-stage renal failure, hemolysis, trauma,

Addison's disease, metabolic acidosis, acute starvation, dehydration, and with rapid K infusion.Chloride is increased in dehydration, renal tubular acidosis (hyperchloremia

metabolic acidosis), acute renal failure, metabolic acidosis associated with prolonged diarrhea and loss of sodium bicarbonate, diabetes insipidus, adrenocortical hyperfuction,

salicylate intoxication and with excessive infusion of isotonic saline or extremely high dietary intake of salt.Chloride is decreased in overhydration, chronic respiratory acidosis,

salt-losing nephritis, metabolic alkalosis, congestive heart failure, Addisonian crisis, certain types of metabolic acidosis, persistent gastric secretion and prolonged vomiting,

URINALYSIS-Routine urine analysis assists in screening and diagnosis of various metabolic, urological, kidney and liver disorders

Protein: Elevated proteins can be an early sign of kidney disease. Urinary protein excretion can also be temporarily elevated by strenuous exercise, orthostatic proteinuria,

dehydration, urinary tract infections and acute illness with fever

Glucose: Uncontrolled diabetes mellitus can lead to presence of glucose in urine. Other causes include pregnancy, hormonal disturbances, liver disease and certain

medications.

Ketones: Uncontrolled diabetes mellitus can lead to presence of ketones in urine. Ketones can also be seen in starvation, frequent vomiting, pregnancy and strenuous

exercise.

Blood: Occult blood can occur in urine as intact erythrocytes or haemoglobin, which can occur in various urological, nephrological and bleeding disorders.

Leukocytes: An increase in leukocytes is an indication of inflammation in urinary tract or kidneys. Most common cause is bacterial urinary tract infection.

Nitrite: Many bacteria give positive results when their number is high. Nitrite concentration during infection increases with length of time the urine specimen is retained in

bladder prior to collection.

pH: The kidneys play an important role in maintaining acid base balance of the body. Conditions of the body producing acidosis/ alkalosis or ingestion of certain type of food

can affect the pH of urine.

Specific gravity: Specific gravity gives an indication of how concentrated the urine is. Increased specific gravity is seen in conditions like dehydration, glycosuria and

proteinuria while decreased specific gravity is seen in excessive fluid intake, renal failure and diabetes insipidus.

Bilirubin: In certain liver diseases such as biliary obstruction or hepatitis, bilirubin gets excreted in urine.


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

Cert. No. MC-2461








TRUST IPD

UID:10727635 REQNO-6397939

IPD-EMERGENCY

IPID-63236/21/1301



Urobilinogen: Positive results are seen in liver diseases like hepatitis and cirrhosis and in cases of hemolytic anemia

**End Of Report**


Dr. Rahul Bhardwaj,MD.

Pathologist




NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

Cert. No. MC-2461





REFERRING DOCTOR :



TRUST IPD

UID:10727635 REQNO-6398686

IPD-ICU(4)

IPID-63236/21/1301



BIO CHEMISTRY

LIVER FUNCTION PROFILE, SERUM

BILIRUBIN, TOTAL 1.24 0.2 - 1.0 mg/dLHigh

METHOD : DIAZO METHOD

BILIRUBIN, DIRECT 0.42 0.0 - 0.2 mg/dLHigh

METHOD : DIAZO METHOD

BILIRUBIN, INDIRECT 0.47 0.1 - 1.0 mg/dL



METHOD : BIURET


METHOD : SPECTROPHOTOMETRY – BROMOCRESOL PURPLE



ALBUMIN/GLOBULIN RATIO 0.3 1.0 - 2.1 RATIOLow


ASPARTATE AMINOTRANSFERASE (AST/SGOT) 23 15 - 37 U/L

METHOD : SPECTROPHOTOMETRIC-IFCC WITH UV WITH PYRIDOXAL-5-PHOSPHATE

ALANINE AMINOTRANSFERASE (ALT/SGPT) 21 < 34.0 U/L


ALKALINE PHOSPHATASE 124 30 - 120 U/LHigh

METHOD : SPECTROPHOTOMETRY, P-NPP (AMP BUFFER)

GAMMA GLUTAMYL TRANSFERASE (GGT) 28 5 - 55 U/L


LACTATE DEHYDROGENASE 342 100 - 190 U/LHigh

METHOD : LACTATE -PYRUVATE

CALCIUM, SERUM

CALCIUM 7.8 8.5 - 10.1 mg/dLLow

METHOD : O-CRESOLPHTHALEIN COMPLEXONE

MAGNESIUM, SERUM

MAGNESIUM 1.5 1.8 - 2.4 mg/dLLow

METHOD : METHYLTHYMOL BLUE

PHOSPHORUS, SERUM

PHOSPHORUS 3.9 2.5 - 4.9 mg/dL

METHOD : PHOSPHOMOLYBDATE UV FORMATION

Interpretation(s)

LIVER FUNCTION PROFILE, SERUM-


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

Cert. No. MC-2461





REFERRING DOCTOR :



TRUST IPD

UID:10727635 REQNO-6398686

IPD-ICU(4)

IPID-63236/21/1301



LIVER FUNCTION PROFILE

Bilirubin is a yellowish pigment found in bile and is a breakdown product of normal heme catabolism. Bilirubin is excreted in bile and urine, and elevated levels may give

yellow discoloration in jaundice.Elevated levels results from increased bilirubin production (eg, hemolysis and ineffective erythropoiesis), decreased bilirubin excretion (eg,

obstruction and hepatitis), and abnormal bilirubinmetabolism (eg, hereditary and neonatal jaundice). Conjugated (direct) bilirubin is elevated more than unconjugated

(indirect) bilirubin in Viral hepatitis, Drug reactions, Alcoholic liver disease Conjugated (direct) bilirubin is also elevated more than unconjugated (indirect) bilirubin when

there is some kind of blockage of the bile ducts like in Gallstones getting into the bile ducts, tumors &Scarring of the bile ducts. Increased unconjugated (indirect) bilirubin

may be a result of Hemolytic or pernicious anemia, Transfusion reaction & a common metabolic condition termed Gilbert syndrome, due to low levels of the enzyme that

attaches sugar molecules to bilirubin.

AST is an enzyme found in various parts of the body. AST is found in the liver, heart, skeletal muscle, kidneys, brain, and red blood cells, and it is commonly measured

clinically as a marker for liver health. AST levels increase during chronic viral hepatitis, blockage of the bile duct, cirrhosis of the liver,liver cancer,kidney failure,hemolytic

anemia,pancreatitis,hemochromatosis. AST levels may also increase after a heart attack or strenuous activity.ALT test measures the amount of this enzyme in the blood.ALT

is found mainly in the liver, but also in smaller amounts in the kidneys,heart,muscles, and pancreas.It is commonly measured as a part of a diagnostic evaluation of

hepatocellular injury, to determine liver health.AST levels increase during acute hepatitis,sometimes due to a viral infection,ischemia to the liver,chronic hepatitis,obstruction

of bile ducts,cirrhosis.

ALP is a protein found in almost all body tissues.Tissues with higher amounts of ALP include the liver,bile ducts and bone.Elevated ALP levels are seen in Biliary obstruction,

Osteoblastic bone tumors, osteomalacia, hepatitis, Hyperparathyroidism, Leukemia, Lymphoma, Paget's disease,Rickets,Sarcoidosis etc. Lower-than-normal ALP levels seen

in Hypophosphatasia,Malnutrition,Protein deficiency,Wilson's disease.GGT is an enzyme found in cell membranes of many tissues mainly in the liver,kidney and pancreas.It is

also found in other tissues including intestine,spleen,heart, brain and seminal vesicles.The highest concentration is in the kidney,but the liver is considered the source of

normal enzyme activity.Serum GGT has been widely used as an index of liver dysfunction.Elevated serum GGT activity can be found in diseases of the liver,biliary system and

pancreas.Conditions that increase serum GGT are obstructive liver disease,high alcohol consumption and use of enzyme-inducing drugs etc.Serum total protein,also known as

total protein,is a biochemical test for measuring the total amount of protein in serum.Protein in the plasma is made up of albumin and globulin.Higher-than-normal levels may

be due to:Chronic inflammation or infection,includingHIV and hepatitis B or C,Multiple myeloma,Waldenstrom's disease.Lower-than-normal levels may be due to:

Agammaglobulinemia,Bleeding (hemorrhage),Burns,Glomerulonephritis,Liverdisease, Malabsorption,Malnutrition,Nephrotic syndrome,Protein-losing enteropathy etc.Human

serum albumin is the most abundant protein in human blood plasma.It is produced in the liver.Albumin constitutes about half of the blood serum protein.Low blood albumin

levels (hypoalbuminemia) can be caused by:Liver disease like cirrhosis of the liver, nephrotic syndrome,protein-losing enteropathy,Burns,hemodilution,increased vascular

permeability or decreased lymphatic clearance,malnutrition and wasting etc

CALCIUM, SERUM-

Commom causes of decreased value of calcium (hypocalcemia) are chronic renal failure, hypomagnesemia and hypoalbuminemia.

Hypercalcemia (increased value of calcium) can be caused by increased intestinal absorbtion (vitamin d intoxication), increased skeletal reasorption (immobilization),

or a combination of mechanisms (primary hyperparathyroidism). Primary hyperparathyroidism and malignancy accounts for 90-95% of all cases of hypercalcemia.

Values of total calcium is affected by serum proteins, particularly albumin thus, latter’s value should be taken into account when interpreting serum calcium

levels. The following regression equation may be helpful.

Corrected total calcium (mg/dl)= total calcium (mg/dl) + 0.8 (4- albumin [g/dl])*

because regression equations vary among group of patients in different physiological and pathological conditions, mathematical corrections are only

approximations. The possible mathematical corrections should be replaced by direct determination of free calcium by ISE (available with srl) a common and

important source of preanalytical error in the measurement of calcium is prolonged torniquet application during sampling. Thus, this along with fist clenching

should be avoided before phlebotomy.

MAGNESIUM, SERUM-

Moderate or severe magnesium deficiency is usually due to losses of magnesium from gastrointestinal tract or kidneys as in vomiting and diarrhoea in former

and alcohol, diabetes mellitus (osmotic diuresis), loop diuretics (furosemide) and aminoglycoside antibiotics in latter.

Symptomatic hypermagnesemia is almost always caused by excessive intake with concomitant renal failure, thereby decreasing the ability of the kidneys to

excrete excess magnesium.

Magnesium concentration in erythrocytes are approximately three times those of serum. Conversion factors for the units used to express magnesium

concentration are:

mg/dl= meq/l x 1.22 = mmol/l x 2.43

PHOSPHORUS, SERUM-

**End Of Report**





NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

Cert. No. MC-2461





REFERRING DOCTOR :



TRUST IPD

UID:10727635 REQNO-6398918

IPD-ICU(4)

IPID-63236/21/1301



BIO CHEMISTRY

CREATINE KINASE (CPK), SERUM

CREATINE KINASE 41 26 - 192 U/L


CREATINE KINASE - MB, SERUM

CREATINE KINASE - MB 42.0 7 - 25 U/LHigh

METHOD : NAC ACTIVATED MODIFIED IFCC METHOD

Interpretation(s)

CREATINE KINASE (CPK), SERUM-

Creatine phosphokinase is an enyme found mainly in heart, brain & skeletal muscle. It has 3 isoenzymes - CPK-1 ( CPK-BB ) mostly found in brain & lungs, CPK-2 ( CPK-MB )

mostly found in heart, CPK-3 ( CPK-MM ) found mostly in skeletal muscle. CPK is usually elevated in Brain tumors, Brain injury (stroke), pulmonary infarction, myocardial

infarction, crush injuries, Rhabdomyolysis, muscular dystrophy, myositis etc.

SPECIALISED CHEMISTRY - HORMONE

* PROCALCITONIN, SERUM

PROCALCITONIN 4.080 0.25 to <0.5(Grey Zone) indicates

that local bacterial infection is

possible. Systemic infection is not

likely.

< 0.5 Minor or No Significant

Systemic Infection

>= 0.5 - < 2.0 Moderate To Severe

Risk Infection

>= 2.0 - < 10.0 High To Severe

Risk Infection

>= 10.0 Systemic Inflammatory

Response To Severe Bacterial

Infection¿

ng/mLHigh

METHOD : CHEMILUMINESCENT MICROPARTICLE IMMUNOASSAY (CMIA)

Interpretation(s)

PROCALCITONIN, SERUM-

Guideline for reference ranges of Procalcitonin

PCT < 0.05 ng/mL Healthy Individual

PCT < 0.50 ng/mL Low risk for progression to severe systemic sepsis

PCT 0.50 ng/mL – < 2.00 ng/mL Moderate risk for progression to severe systemic sepsis

PCT 2.00 ng/mL - 10.00 ng/mL High risk for progression to severe systemic sepsis

PCT > 10.00 ng/mL High likelihood of severe sepsis or septic shock

References:

Chiesa C et al. Clin Infect Dis 1998, 26: 664-672

Chiesa C et al. Clin Chem 2003, 49(1): 60-68

Joram N et al. Eur J Clin Microbiol Inf Dis 2011, Feb 12


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

Cert. No. MC-2461





REFERRING DOCTOR :



TRUST IPD

UID:10727635 REQNO-6398918

IPD-ICU(4)

IPID-63236/21/1301



**End Of Report**


TEST MARKED WITH '*' ARE OUTSIDE THE NABL ACCREDITED SCOPE OF THE LABORATORY.




NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

Cert. No. MC-2461





REFERRING DOCTOR : SELF



TRUST IPD

UID:10727635 REQNO-6398952

IPD-ICU(4)

IPID-63236/21/1301



BIO CHEMISTRY

C-REACTIVE PROTEIN, SERUM

C-REACTIVE PROTEIN 191.2 < 10.0 mg/LHigh

SPECIALISED CHEMISTRY - ANEMIA

FERRITIN, SERUM

FERRITIN 215.2 4.63 - 204.00 ng/mLHigh

METHOD : CHEMILUMINESCENT MICROPARTICLE IMMUNOASSAY (CMIA)

Interpretation(s)

FERRITIN, SERUM-Ferritin is a high-molecular-weight protein that contains approximately 20% iron. It occurs normally in almost all tissues of the body but especially in

hepatocytes and reticuloendothelial cells, where it serves as an iron reserve. When needed, the iron molecules are released from the apoferritin shell and bind to transferrin,

the circulating plasma protein that transports iron to the erythropoietic cells.

A low serum ferritin value is thought to be the best laboratory indicator of iron depletion. Virtually all patients with low serum iron and low ferritin have iron deficiency. Serum

Ferritin concentration, when considered with other factors such as serum iron, iron-binding capacity and tissue iron stores is valuable in the diagnosis of iron deficiency

anemia, anemia of chronic infection and conditions such as thalassemia and hemochromatosis that are associated with iron overload. It is particularly useful in distinguishing

between iron-deficiency anemia (serum ferritin levels diminished) and "anemia of chronic disease" (serum ferritin levels usually normal or elevated).

Ferritin is an acute phase reactant. It can be found to be elevated in the following conditions and do not reflect actual body iron stores: 1.Inflammation 2.Significant tissue

destruction 3.Liver diseases 4.Malignancies such as acute leukemia and Hodgkin's disease 5.Therapy with iron supplements.

Interferences:

Heterophilic antibodies in human serum can react with reagent immunoglobulins, interfering with in vitro immunoassays. Patients routinely exposed to animals or to animal

serum products can be prone to this interference and anomalous values may be observed.

**End Of Report**





NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED








TRUST IPD

UID:10727635 REQNO-6398952

IPD-ICU(4)

IPID-63236/21/1301



ENDOCRINOLOGY

INTERLEUKIN 6, SERUM

INTERLEUKIN 6 93.20 <3.4 pg/mLHigh

Interpretation(s)

INTERLEUKIN 6, SERUM-

Interleukin-6 (IL-6) is a pleiotropic cytokine with a wide range of functions, IL-6 is produced from a single gene encoding a product of 212 amino acids, which is cleaved at

the N-terminus to produce a 184 amino acid peptide with a molecular weight between 22-27 kDa.

IL-6 production is rapidly induced in the course of acute inflammatory reactions associated with injury, trauma, stress, infection, brain death, neoplasia and other situations.

IL-6 concentrations in trauma patients may predict later complications from additional surgical stress or indicate missed injuries or complications.

Sequential measurements of IL-6 in serum or plasma of patients admitted to the ICU (intensive care unit) showed to be useful in evaluating the severity of SIRS (systemic

inflammatory response syndrome), sepsis and septic shock and to predict the outcome of these patients. IL-6 is also useful as an early alarm marker for the detection of

neonatal sepsis.IL-6 also plays a role in chronic inflammation e.g. rheumatoid arthritis.

**End Of Report**


Dr. Shakti Aggarwal

Senior Biochemist

Dr. Anurag Bansal

LAB DIRECTOR

Page 1 Of 1SRL,REFERENCE LAB, GP-26, MARUTI INDUSTRIAL ESTATE,UDYOG VIHAR,SECTOR-18,

GURGAON, 122015

HARYANA, INDIA

Tel : 9111591115, CIN - U74899PB1995PLC045956

Email : [email protected]

SRL LIMITEDSRL,REFERENCE LAB, GP-26, MARUTI INDUSTRIAL ESTATE,UDYOG VIHAR,SECTOR-18,

GURGAON, 122015

HARYANA, INDIA

Tel : 9111591115, CIN - U74899PB1995PLC045956


SRL LIMITED








TRUST IPD

ICMR Registration No: SRLRL001

UID:10727635 REQNO-6401536

IPD-ICU(4)

IPID-63236/21/1301



MOLECULAR BIOLOGY

SARS COV -2 REAL TIME PCR

SARS-COV-2 RNA NEGATIVE

Interpretation(s)

SARS COV -2 REAL TIME PCR-

SARS-CoV-2, formerly known as 2019-nCoV, is the causative agent of the coronavirus disease 2019 (COVID-19). Main symptoms of the disease include fever, cough and

shortness of breath. The virus is spread via person-to-person contact through respiratory droplets produced when a person coughs or sneezes. The SARS-CoV-2 RNA is

generally detectable in nasopharyngeal/oropharyngeal swabs during the acute phase of infection. Positive results are indicative of active infection. Real Time PCR assay

targets specific genes and can be used for diagnosis of SARS-CoV-2 virus infection which contributes to severe upper respiratory distress and complications.

Positive result indicates that RNA from SARS-CoV-2 was detected in the specimen, and the patient is considered infected with the virus and presumed to be contagious.

Negative test result for this test means that SARS-CoV-2 RNA was not detected in the specimen above the limit of detection of the assay.

Limitations:

• Negative results do not preclude COVID-19 and should not be used as the sole basis for patient management decisions. Negative results must be combined with clinical

observations, patient history, and epidemiological information.

• Positive results do not rule out bacterial infection or co-infection with other viruses.

• Optimum specimen types and timing for peak viral levels during infections caused by 2019-nCoV have not been determined. Collection of multiple specimens (types and

time points) from the same patient may be necessary to detect the virus.

• Follow-up testing may particularly be important if patient has a clinical picture of viral pneumonia, a potential exposure history, and/or radiographic findings (chest CT or

MRI scan) consistent with COVID -19 pneumonia. However repeat testing in the near-term after clearance (within 90 days) should be avoided as prolonged shedding of

non-viable virus is not uncommon

• Ct values generated from different assay systems within the same laboratory, or from different laboratories, are not directly comparable and do not necessarily reflect the

same viral load due to inter-assay and inter-laboratory variability.

• Variation in timing of sample collection, fluctuations in virus shedding, and difference between detection limit of different testing methods within same or different labs could

lead to variation in results particularly during initial phase of infection.

• If the virus mutates in the rRT-PCR target region, 2019-nCoV may not be detected or may be detected less predictably. Inhibitors or other types of interference may

produce a false negative result.

• The performance of this test has not been established for monitoring treatment of 2019-nCoV infection.

Note: Test is performed using ICMR approved Kit.

References:

1. Laboratory testing for coronavirus disease 2019 (COVID-19) in suspected human cases. Interim guidance. World Health Organization.

2. Druce et al. JCM. 2011

3. N. Engl. J. Med. 2020, 382, 929–936

**End Of Report**


Dr. Anurag Bansal

LAB DIRECTOR

Dr. Rashmi Talwar, PhD

Section Head- Genetics

Dr. Yoginder Pal Singh, Ph.D

Molecular Biologist

Page 1 Of 1SRL,REFERENCE LAB, GP-26, MARUTI INDUSTRIAL ESTATE,UDYOG VIHAR,SECTOR-18,

GURGAON, 122015

HARYANA, INDIA

Tel : 9111591115, CIN - U74899PB1995PLC045956


SRL LIMITEDSRL,REFERENCE LAB, GP-26, MARUTI INDUSTRIAL ESTATE,UDYOG VIHAR,SECTOR-18,

GURGAON, 122015

HARYANA, INDIA

Tel : 9111591115, CIN - U74899PB1995PLC045956


SRL LIMITED

Cert. No. MC-2461





REFERRING DOCTOR :



TRUST IPD

UID:10727635 REQNO-6406271

IPD-ICU(4)

IPID-63236/21/1301



KIDNEY PANEL - 1

SERUM BLOOD UREA NITROGEN

BLOOD UREA NITROGEN 17 6 - 20 mg/dL

METHOD : UREASE -GLDH

CREATININE EGFR- EPI

CREATININE 0.67 0.60 - 1.10 mg/dL

METHOD : MODIFIED JAFFE KINETIC

AGE 59 years

METHOD : MANUAL

GLOMERULAR FILTRATION RATE (FEMALE) 96.21 Refer Interpretation Below mL/min/1.73m2


BUN/CREAT RATIO

BUN/CREAT RATIO 25.37 5.00 - 15.00High


URIC ACID, SERUM

URIC ACID 3.6 2.6 - 6.0 mg/dL

METHOD : URICASE/ PAP

TOTAL PROTEIN, SERUM


METHOD : BIURET

Comments

NOTE :- KINDLY CORRELATE CLINICALLY.

ALBUMIN, SERUM


METHOD : BROMOCRESOL PURPLE

GLOBULIN



ELECTROLYTES (NA/K/CL), SERUM

SODIUM 140 136 - 145 mmol/L


POTASSIUM 4.20 3.50 - 5.10 mmol/L


CHLORIDE 104 98 - 107 mmol/L


URINALYSIS

COLOR PALE YELLOW

METHOD : MANUAL


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

Cert. No. MC-2461





REFERRING DOCTOR :



TRUST IPD

UID:10727635 REQNO-6406271

IPD-ICU(4)

IPID-63236/21/1301



APPEARANCE HAZY

METHOD : MANUAL

PH 5.5 4.7 - 7.5

METHOD : DIPSTICK

SPECIFIC GRAVITY 1.030 1.003 - 1.035

METHOD : DIPSTICK

GLUCOSE DETECTED (+++) NOT DETECTED


PROTEIN DETECTED (+) NOT DETECTED


KETONES DETECTED (TRACE) NOT DETECTED


BLOOD DETECTED (+) IN

URINE

NOT DETECTED

METHOD : DIPSTICK

BILIRUBIN NOT DETECTED NOT DETECTED


UROBILINOGEN NORMAL NORMAL


NITRITE NOT DETECTED NOT DETECTED

METHOD : DIPSTICK

WBC 8-10 0-5 /HPF


EPITHELIAL CELLS 2-3 0-5 /HPF


RED BLOOD CELLS 3 - 5 NOT DETECTED /HPF


CASTS GRANULAR CAST SEEN.


CRYSTALS NOT DETECTED


Comments

NOTE :- MICROSCOPIC EXAMINATION OF URINE IS PERFORMED BY CENTRIFUGED URINARY SEDIMENT.

NOTE :- KETONE IN URINE IN ABSENCE OF GLYCOSURIA MAY BE FOUND IN CASES OF

FEVER, VOMITING, DEHYDRATION ETC.

Interpretation(s)

SERUM BLOOD UREA NITROGEN-


Pre renal

• High protein diet, Increased protein catabolism, GI haemorrhage, Cortisol, Dehydration, CHF Renal

• Renal Failure


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

Cert. No. MC-2461





REFERRING DOCTOR :



TRUST IPD

UID:10727635 REQNO-6406271

IPD-ICU(4)

IPID-63236/21/1301



Post Renal

• Malignancy, Nephrolithiasis, Prostatism


• Liver disease

• SIADH.

CREATININE EGFR- EPI-

GFR— Glomerular filtration rate (GFR) is a measure of the function of the kidneys. The GFR is a calculation based on a serum creatinine test. Creatinine is a muscle waste

product that is filtered from the blood by the kidneys and excreted into urine at a relatively steady rate. When kidney function decreases, less creatinine is excreted and

concentrations increase in the blood. With the creatinine test, a reasonable estimate of the actual GFR can be determined.

A GFR of 60 or higher is in the normal range.

A GFR below 60 may mean kidney disease.

A GFR of 15 or lower may mean kidney failure.

Estimated GFR (eGFR) is the preferred method for identifying people with chronic kidney disease (CKD). In adults, eGFR calculated using the Modification of Diet in Renal

Disease (MDRD) Study equation provides a more clinically useful measure of kidney function than serum creatinine alone.

The CKD-EPI creatinine equation is based on the same four variables as the MDRD Study equation, but uses a 2-slope spline to model the relationship between estimated GFR

and serum creatinine, and a different relationship for age, sex and race. The equation was reported to perform better and with less bias than the MDRD Study equation,

especially in patients with higher GFR. This results in reduced misclassification of CKD.

The CKD-EPI creatinine equation has not been validated in children & will only be reported for patients = 18 years of age. For pediatric and childrens, Schwartz Pediatric

Bedside eGFR (2009) formulae is used. This revised "bedside" pediatric eGFR requires only serum creatinine and height.

URIC ACID, SERUM-


Dietary

• High Protein Intake.

• Prolonged Fasting,

• Rapid weight loss.

Gout

Lesch nyhan syndrome.

Type 2 DM.

Metabolic syndrome.


• Low Zinc Intake

• OCP’s

• Multiple Sclerosis

Nutritional tips to manage increased Uric acid levels

• Drink plenty of fluids

• Limit animal proteins

• High Fibre foods

• Vit C Intake

• Antioxidant rich foods

TOTAL PROTEIN, SERUM-

Serum total protein,also known as total protein, is a biochemical test for measuring the total amount of protein in serum..Protein in the plasma is made up of albumin and

globulin

Higher-than-normal levels may be due to: Chronic inflammation or infection, including HIV and hepatitis B or C, Multiple myeloma, Waldenstrom's disease

Lower-than-normal levels may be due to: Agammaglobulinemia, Bleeding (hemorrhage),Burns,Glomerulonephritis, Liver disease, Malabsorption, Malnutrition, Nephrotic

syndrome,Protein-losing enteropathy etc.

ALBUMIN, SERUM-

Human serum albumin is the most abundant protein in human blood plasma. It is produced in the liver. Albumin constitutes about half of the blood serum protein. Low blood

albumin levels (hypoalbuminemia) can be caused by: Liver disease like cirrhosis of the liver, nephrotic syndrome, protein-losing enteropathy, Burns, hemodilution, increased

vascular permeability or decreased lymphatic clearance,malnutrition and wasting etc.

ELECTROLYTES (NA/K/CL), SERUM-

Sodium levels are Increased in dehydration, cushing's syndrome, aldosteronism & decreased in Addison's disease, hypopituitarism,liver disease. Hypokalemia (low K) is

common in vomiting, diarrhea, alcoholism, folic acid deficiency and primary aldosteronism. Hyperkalemia may be seen in end-stage renal failure, hemolysis, trauma,

Addison's disease, metabolic acidosis, acute starvation, dehydration, and with rapid K infusion.Chloride is increased in dehydration, renal tubular acidosis (hyperchloremia

metabolic acidosis), acute renal failure, metabolic acidosis associated with prolonged diarrhea and loss of sodium bicarbonate, diabetes insipidus, adrenocortical hyperfuction,

salicylate intoxication and with excessive infusion of isotonic saline or extremely high dietary intake of salt.Chloride is decreased in overhydration, chronic respiratory acidosis,

salt-losing nephritis, metabolic alkalosis, congestive heart failure, Addisonian crisis, certain types of metabolic acidosis, persistent gastric secretion and prolonged vomiting,

URINALYSIS-Routine urine analysis assists in screening and diagnosis of various metabolic, urological, kidney and liver disorders

Protein: Elevated proteins can be an early sign of kidney disease. Urinary protein excretion can also be temporarily elevated by strenuous exercise, orthostatic proteinuria,

dehydration, urinary tract infections and acute illness with fever

Glucose: Uncontrolled diabetes mellitus can lead to presence of glucose in urine. Other causes include pregnancy, hormonal disturbances, liver disease and certain

medications.

Ketones: Uncontrolled diabetes mellitus can lead to presence of ketones in urine. Ketones can also be seen in starvation, frequent vomiting, pregnancy and strenuous

exercise.

Blood: Occult blood can occur in urine as intact erythrocytes or haemoglobin, which can occur in various urological, nephrological and bleeding disorders.


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

Cert. No. MC-2461





REFERRING DOCTOR :



TRUST IPD

UID:10727635 REQNO-6406271

IPD-ICU(4)

IPID-63236/21/1301



Leukocytes: An increase in leukocytes is an indication of inflammation in urinary tract or kidneys. Most common cause is bacterial urinary tract infection.

Nitrite: Many bacteria give positive results when their number is high. Nitrite concentration during infection increases with length of time the urine specimen is retained in

bladder prior to collection.

pH: The kidneys play an important role in maintaining acid base balance of the body. Conditions of the body producing acidosis/ alkalosis or ingestion of certain type of food

can affect the pH of urine.

Specific gravity: Specific gravity gives an indication of how concentrated the urine is. Increased specific gravity is seen in conditions like dehydration, glycosuria and

proteinuria while decreased specific gravity is seen in excessive fluid intake, renal failure and diabetes insipidus.

Bilirubin: In certain liver diseases such as biliary obstruction or hepatitis, bilirubin gets excreted in urine.

Urobilinogen: Positive results are seen in liver diseases like hepatitis and cirrhosis and in cases of hemolytic anemia

HAEMATOLOGY


BLOOD/SMEAR

BLOOD COUNTS

HEMOGLOBIN 10.5 12.0 - 15.0 g/dLLow




WHITE BLOOD CELL COUNT 11.09 4.0 - 10.0 thou/µLHigh





HEMATOCRIT 32.55 36 - 46 %Low

METHOD : MEASURED


METHOD : MEASURED




CONCENTRATION

32.2 31.5 - 34.5 g/dL









ABSOLUTE NEUTROPHIL COUNT 10.20 2.0 - 7.0 thou/µLHigh





NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

Cert. No. MC-2461





REFERRING DOCTOR :



TRUST IPD

UID:10727635 REQNO-6406271

IPD-ICU(4)

IPID-63236/21/1301












ABSOLUTE MONOCYTE COUNT 0.33 0.2 - 1.0 thou/µL








Interpretation(s)

BLOOD COUNTS-









show mild disease.



**End Of Report**


Dr. Rahul Bhardwaj,MD.

Pathologist




NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

Cert. No. MC-2461





REFERRING DOCTOR :



TRUST IPD

UID:10727635 REQNO-6414176

IPD-ICU(2)

IPID-63236/21/1301



HAEMATOLOGY


BLOOD/SMEAR

BLOOD COUNTS

HEMOGLOBIN 10.0 12.0 - 15.0 g/dLLow




WHITE BLOOD CELL COUNT 6.47 4.0 - 10.0 thou/µL




Comments

GIANT PLATELETS SEEN.


HEMATOCRIT 30.61 36 - 46 %Low

METHOD : MEASURED


METHOD : MEASURED




CONCENTRATION

32.5 31.5 - 34.5 g/dL









ABSOLUTE NEUTROPHIL COUNT 5.69 2.0 - 7.0 thou/µL








NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

Cert. No. MC-2461





REFERRING DOCTOR :



TRUST IPD

UID:10727635 REQNO-6414176

IPD-ICU(2)

IPID-63236/21/1301









ABSOLUTE MONOCYTE COUNT 0.19 0.2 - 1.0 thou/µLLow








Interpretation(s)

BLOOD COUNTS-









show mild disease.



BIO CHEMISTRY

POTASSIUM, SERUM

POTASSIUM 3.60 3.50 - 5.10 mmol/L


SODIUM, SERUM

SODIUM 142 136 - 145 mmol/L


Interpretation(s)

POTASSIUM, SERUM-

Hypokalemia (low K) is common in vomiting, diarrhea, alcoholism, folic acid deficiency and primary aldosteronism. Hyperkalemia may be seen in end-stage renal failure,

hemolysis, trauma, Addison's disease, metabolic acidosis, acute starvation, dehydration, and with rapid K infusion.

SODIUM, SERUM-

Increased in dehydration, cushing's syndrome, aldosteronism; Decreased in Addison's disease, hypopituitarism,liver disease.


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

Cert. No. MC-2461





REFERRING DOCTOR :



TRUST IPD

UID:10727635 REQNO-6414176

IPD-ICU(2)

IPID-63236/21/1301



**End Of Report**





NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

Cert. No. MC-2461





REFERRING DOCTOR :



TRUST IPD

UID:10727635 REQNO-6417320

IPD-ICU(2)

IPID-63236/21/1301



MICRO BIOLOGY

GRAM STAIN

SPECIMEN SOURCE BRONCHOALVEOLAR LAVAGE (MINI)


GRAM STAIN SMEAR SHOWS FEW PUS CELLS, NO MICRO-ORGANISMS SEEN.


**End Of Report**


Dr.Rahul Behl,M.D.

Microbiologist


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956


NEW DELHI, 110070

DELHI, INDIA

Tel : 011-42776222,

CIN - U74899PB1995PLC045956

SRL LIMITED

PATIENT NAME : MRS. KIRAN SHARMA - GiveIndia

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