Muntah Dan Kembung Pada Bayi Dan Anak

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    Muntah dan KembungPada Bayi dan Anak

    ( Pendekatan Klinis )

    Kustiyo Gunawan

    FK Unair Surabaya

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    Bilious Vomiting in the Newborn: How Often Is It Pathologic?By Prasad Godbole and Mark D. Stringer

    Leeds, EnglandJ Pediatr Surg 37:909-911. Copyright 2002, Elsevier Science

    (USA).

    Conclusions:These data emphasize the maxim that bilious

    vomiting in the newborn should be attributed to intestinal

    obstruction until proved otherwise. However, in this prospective

    audit, bilious vomiting was not caused by intestinalobstruction in 62% of cases, and most of these infants suffered

    no further sequelae.

    Intest inal malrot at ion must be excluded

    specifically.

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    Vomiting, Regurgitation, Rumination

    Vomiting : Keluarnya isi lambung/usus dari mulut secara

    sadar disertai kontraksi dinding abdomen

    Regurgitation: Keluarnya makanan tanpa tenaga, biasanyaberasal dari esofagus (pseudovomiting)

    Rumination : Secara sadar dan otomatis membawa makanan

    ke mulut dikunyahditelandibawa ulang ke

    mulut

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    Kausa Mutah

    Iritasi ujung saraf di peritoneum Obstruksi organ berrongga

    atau mesenterium (intestine, ren, ureter, uterin canal,

    vermiform appendix)

    Gangrenous appendix Peristaltic contractionAcute Pancreatitis

    Strangulation

    Torsion of the pedicle of an ovarian cyst Stretching of the muscular

    wall/spasm

    Pain (colic)

    Vomiting (occurs at the height

    of the spasm)

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    Relationship of Vomiting

    Pain Frequency of Character of Nausea & loss of Others

    The Vomiting The Vomiting Appetite symptoms

    (no vomit)

    Vomiting Directly with Duod. atr

    Comming acuteness Delayed

    after pain (appendicitis, H P S passages

    (appendicitis) pancreatitis) of meconium

    Int. obstr

    Vomiting early, obstr.of the same st imulus

    Sudden & violent colon no vomit dif ferent grade

    (acute obst.ureter if vomit,

    /Bile duct) incomp. IC valve

    Vomiting after pain, Acute loss of Nausea Vomiting

    depend on how high appetite &

    of intestinal obst. pain

    (appendicitis)

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    Vomiting (Prompt Dx and Tx)

    Abdominal Not Surgical indoubt indefinite

    Emergencies symptoms

    Extra GI origin GI origin

    (inf.of the gut) Discuss

    Bilious Psychogenic w a i t (discuss)CT,US

    Persistent Infectious pathology

    Acute loss of appetite Neuromeningeal inf

    Blood Metabolic pathology improve not improve judge

    Pain Intoxication

    observes

    Urgent need operation

    Delayed Transfer correct diagnosis

    Short gut survive

    syndrome urgent need for operation

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    Importance of evaluating for cow's milk allergy in pediatricsurgical patients with functional bowel symptoms

    Kayo Ikedaa, Shinobu Idaa, Hisayoshi Kawaharab,., 1, Koji Kawamotoa,

    Yuri Etania, Akio Kubotab

    Journal of Pediatric Surgery (2011) 46, 23322335

    Conclusions: A high index of suspicion regarding

    the possibility of concurrent CMA

    may be necessary to manage bowel

    symptoms in pediatric surgicalpatients.

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    I L E U SIntestinal dilatation, Hypersecretion, bacterial overgrowth

    Mechanical obstruction Non mechanical obstruction

    (extrinsic/intrinsic) (GI paralysis, pseudo-obstruction)

    Acute/Chronis Partial/Complete Simple / Closed loop

    (Traps the bowel mesentery)

    Intraabdominal adhesion Blood supply compromise

    Stricture

    Tumor Ischemia

    Congenital

    Necrosis

    Perforation

    S t r a n g u l a t i o n

    Hernia, adhesion, volvulus

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    HISTORY AND CLINICAL SETTINGCC: Acute obstipation, Abdominal pain, Distention, Nausea, Vomiting

    Mechanical obstruction PseudoobstructionPain location : Middle of the abdomen Diffuse of the abdomen

    Pain severity : Severe Mild

    Pain character : Increase severity Increase severity

    and depth overtime and depth overtime

    Pain decrease (fatique)

    Proximal Int. Obst. : Short periodically 3 4Periodicity of Pain

    Distal small /large bowel: 15 - 20

    Pain Abdominal distention_____________________________

    Nausea, vomiting, cramping

    Sudden Progressive partial

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    CAUSE AND TYPE OF BOWEL OBSTRUCTION

    In Hospital

    Medical History Medical History & Courses

    Previous episodes of Medication:

    Bowel obst: anticoagulants

    etiology ?

    chemotherapyresponse of Tx ? Metabolic

    Previous of abd/pelvic Abdominal radiation

    Operation: Severe infection

    Operative report Fluid & elect imbalance

    History of malignancy Narcotic recurrence ? Intraabd. inflammation

    History of intraabdominal

    inflammation

    @ A b d o m i n a l D i s t e n t i o n

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    A b d o m i n a l P a i n

    Abdominal Distention, Nausea, Vomiting

    Gradual change in bowel Developing many Minimal crampy abd. In hospital

    Habit weeks Nausea

    Progressive abdominal Vomiting

    DistentionMild /crampy pain after Longstanding intermittent

    Meals mechanical obstruction

    Weight loss

    Chronic Partial Mechanical Chronic process/ Chronic Intestinal Gastric atony

    Bowel Obstruction Progressive Partial Pseudo Obstruction Smallbowel Ileus

    Bowel Obstruction (CIPO) Acute Colonic

    Last flatus Pseudoobstr.

    Partial @ Complete

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    Cows milk protein allergy presenting with Hirschsprungsdiseasemimicking symptomsAkio Kubotaa,*, Hisayoshi Kawaharaa, Hiroomi Okuyamaa, Yoshiyuki Shimizua,

    Mariko Nakachob, Shinobu Idab, Masahiro Nakayamac, Akira Okadaa

    Journal of Pediatric Surgery (2006) 41, 2056 2058

    Conclusion: The proportion of CMA in the cases presenting with

    HD-like symptoms in the neonatal period is much

    higher than what we expected, and most cases of

    BTNIN (benign transient nonorganic ileus of Neonates) are

    caused by CMA. If HD is ruled out, CMA

    should be considered.

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    Whole intestinal pattern on plain abdominal x-

    ray. Both the colon and small intestine are

    markedly dilated. A few air-fluid levels are

    observed.

    Contrast enema. The size of the rectum and colon

    is normal, and no caliber change is observed, but

    the rectum and ascending colon demonstrate

    irregularity of the wall.

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    PHYSICAL EXAMINATION AND RESUSCITATION

    Sense of the Px illness INITIAL STEP Assessing the Px vital sign,

    & course hydration status, CP system

    Volume ? Clear

    NG Tube Bilious non feculent

    Physical Examination Character - Prox. SBO

    -Colonic obst+comp. IC valve

    Volume Feculent

    Urine -Distal SBO

    Character

    IV line: water & electrolyte replacement

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    Degree of distention: Prox little/no distention

    Scar

    O b s e r v a t i o n Malignancy

    Asymetri AbscessClosed loop

    Peristaltic waves: acute SBO

    High pitch + rush + crumpy pain: Obstructive process

    A u s c u l t a t i o n Bowel sound Intestinal paralysis

    Intestinal fatique Longstanding

    obstruction

    Closed loop

    obstruction

    Guarding

    P a l p a t i o n Rebound tenderness Strangulation

    Localized tenderness

    P e r c u s s i o n Dullnessmass

    Tympani distended bowel

    Rectum : Mass ? Fecal impaction ? Occult blood ?

    T o u c h e r

    Ileostomy : Exam stoma ( obstruction at the level of the stoma )

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    K e s i m p u l a n:

    Tidak ada pemeriksaan tambahan sebelum pertimbangan klinis

    Anamnesis dan pemeriksaan fisik harus terstrukturDiagnosis dan penangana dini

    Keterlambatan : kematian / sequele