Why Focusing on Nurture Made and Still Makes Sense: The Biosocial Development of Self-Control
Transcript of Why Focusing on Nurture Made and Still Makes Sense: The Biosocial Development of Self-Control
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Why Focusing on Nurture Made and Still Makes Sense:
The Biosocial Development of Self-Control
Alexander T. Vazsonyi and Joshua W. Roberts
University of Kentucky
Li Huang
Tuskegee University
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Introduction
In the current chapter, we examine both theoretical and subsequent empirical work on
how self-control develops in children based on Gottfredson and Hirschi’s (1990) General Theory
or Crime. Few longitudinal studies (e.g., Polakowski, 1994; Vazsonyi and Huang, 2010; cf., for
cross-sectional tests, Hay, 2001; Perrone, Sullivan, Pratt, and Margaryan, 2004) have addressed
perhaps one of the most pivotal theoretical arguments, and one of self-control theory’s basic
tenets, namely that early socialization processes during the first decade of a child’s life
effectively send a child on a life-long self-control trajectory, one highly predictive of norm
violations, of deviance, and of criminal conduct. Consider the following analogy:
An adolescent who is an incredible baseball pitcher (3 or more standard deviations above a population mean in terms of speed, accuracy and so forth) will be situated in a similar position relative to his peers at 20, 30, 40, and age 50, following a natural trajectory of improved pitching prowess during late adolescents into young adulthood, followed by an inevitable decline. The same logic applies to deviance and crime, considering low self-control. It is the inevitable decline that puzzles social and behavioral scientists, not only in crime and deviance, but a whole host of other observable behaviors (e.g., Kanazawa, 2003; “genius curve”). So, perhaps we should be most interested in developments during the first decade of life and less so in the immutable, universal, maturational growth and decline that follows (Vazsonyi and Huang, 2010: 255).
Despite a large number of empirical tests focused on self-control theory, few have tackled
this issue and central question of how self-control develops, not the exclusive source of
variability in deviance, but a central, probabilistic one. Most related work in criminology is
focused on the stability of self-control (the stability thesis); this only addresses a very modest
piece of the puzzle related to the development of self-control. Some of this (construct) stability
work found evidence supporting theoretical predictions about rank order changes over time
(Arneklev, Cochran, & Gainey, 1998; Hay & Forrest, 2006; Turner & Piquero, 2002), while
others did not (Burt, Simons, & Simons, 2006; Mitchell & MacKenzie, 2006; Winfree, Taylor,
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He, & Esbensen, 2006).
On the other hand, some researchers in the developmental sciences have focused on how
self-control/self-regulation develops in infants and young children for some time (Kopp, 1982);
this includes an understanding that this process was reciprocal, although most of this work was
conducted recently (Colman, Hardy, Albert, Raffaelli, & Crockett, 2006; Kochanska, Coy, &
Murray, 2001; Kochanska, Aksan, Penney, & Doobay, 2007; Raffaelli, Crockett, & Shen, 2005;
Rothbart, Ellis, Rueda, & Posner, 2003). Other developmental work has also focused on the
association between self-control or self-regulation and deviance or externalizing behaviors,
inspired by the work of Mischel and colleagues (1989; Eisenberg, Zhou, Spinrad, Valiente,
Fabes, & Liew, 2005; Zhou, Hofer, Eisenberg, Reiser, Spinrad, & Fabes, 2007; see also Brody &
Ge, 2001; Crockett, Raffaelli, & Shen, 2006). For instance, Eisenberg and colleagues (2005) also
identified how socialization experiences from caregivers continue to contribute in developmental
changes of self-control or self-regulation during childhood and adolescence. They recognized
some time ago that different terminology and at times operationalizations of self-control and
self-regulation across different disciplines or sub-disciplines within social and behavioral
sciences have been used, thus further contributing to how much or little scholarship in this area,
including on self-control theory, has progressed (Eisenberg, Champion, and Ma, 2004).
In the current chapter, we examine the extent to which Gottfredson and Hirschi’s self-
control theory was mis-specified, as some have recently argued, in light of recent scholarship
focused on biological origins of self-control, of deviance, antisocial or criminal behaviors --
some based on the behavior genetic method, some focused on alleles frequency or genetic
markers, and some focused on gene X environment interactions. For instance, Wright and Beaver
(2005) suggest Gottfredson and Hirschi (1990: 60) explicitly discount the possibility that low
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self-control might have a genetic component: “the magnitude of the ‘genetic effect’” they say
[omitted by authors: as determined by adoption studies], “is near zero.” A large body of
literature, however, has arrived at a very different conclusion.
The statement by Wright and Beaver is persuasive; however, Gottfredson and Hirschi
(1990: 60) in fact argue the opposite, on the very same page, commenting most generally on
biological positivism, on searches for physiognomy in criminals, on chromosal markers and so
forth, “This result should not be surprising, and it should not be interpreted as that biology has
nothing to do with crime [emphasis added]”. Recognizing and understanding the behavior
genetic method prior to the publication of their seminal book, almost a quarter century ago, and
undoubtedly due to regular collegial exchanges with David C. Rowe at the University of Arizona
at the time, they also comment on the following page that:
“These correlation coefficients [path model decomposition of a genetic effect on crime shown on page 61] translate into a heritability coefficient of .177, which may be described as the theoretical upper limit of the correlation between father’s criminality and son’s criminality given our assumptions. We would not argue that this result proves the irrelevance of biology to crime. We would argue, however, that this shows the need for greater attention to conceptual matters by those interested in biology and crime” (Gottfredson and Hirschi, 1990: 61).
These comments appear to be mostly focused on conceptual issues, on positivism in general
which is characterized by a data driven, reductionist approach that often lacks an idea, a
conceptual framework, or even a theory. As is often the case, with controversial conceptual or
theoretical work, work that apparently has inspired a generation of criminologists judging by the
sheer volume of scholarship it has generated, work that challenges accepted or agreed upon
notions of how crime and deviance develop, but also work that is simply profound and
parsimonious, as is the case with self-control theory, it precipitates heavy criticism.
It is clear that Gottfredson and Hirschi favored social explanations in how self-control
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develops, but very critically, they repeatedly acknowledged and discussed the salience of both
individual differences, the importance of biology, and of socialization effects. Thus, the current
chapter is guided by the simple question to what extent did Gottfredson and Hirschi (1990) “have
it right” a quarter century ago? Does the General Theory leave out or even ignore biology in the
development of self-control and in the understanding of deviance and crime, as some have levied
as a criticism against the theory? How does self-control develop, using a biosocial lens? We
attempt to address these questions, by providing a larger framework for the development of self-
control, and also, by providing a simple empirical test at the end of the chapter. This is not meant
to fully address our original questions, but simply an illustration of the importance of a reciprocal
socialization process known to underlie the development of self-control and the associated
likelihood of engaging in crime and deviance – an illustration (not in a genetically informed
design) of reciprocity in socialization, evidence for both individual differences as well as
socialization effects in the development of self-control and deviance.
On the Development of Self-Control and the Importance of Biology
Few would question the impact Gottfredson and Hirschi’s (1990) self-control theory has
had on our understanding of crime and deviance within criminology; this is also true outside of
criminology, sometimes explicitly linked to self-control theory and sometimes not, in
psychology (e.g., Baumeister, Heatherton & Tice, 1994; Tangney, Baumeister, & Boone, 2004),
the developmental sciences (Eisenberg et al, 2005), educational sciences (Duckworth &
Seligman, 2005), or health and health-risk research (e.g., Griffin, Scheier, Acevedo, Grenard,
and Botvin, 2011; Miller, Barnes, & Beaver, 2011). Given the temporal order, it seems likely
that Gottfredson and Hirschi’s General Theory might have been an important source, something
DeLisi (2013) so eloquently identifies as a “transdisciplinary” phenomenon in his recent chapter
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entitled “Pandora’s Box.” The extent to which this phenomenon is truly transdisciplinary versus
simply a phenomenon appearing across a number of subdisciplines within social and behavioral
sciences is open for discussion. Gottfredson (2006: 96) so simply, but poignantly noted:
If theories may be judged by how much research they stimulate, control theory is doing exceptionally well. If theories may be judged by their consistency with the facts, control theory is doing exceptionally well. And if theories may be judged by the frequency with which other perspectives seek to incorporate them, control theory is, perhaps, without peer.
Despite its success in generating a tremendous amount of scholarship, the theory has not
been without heavy criticisms and has generated a tremendous amount of debate (Geiss, 2000;
Goode, 2008). Among others, important criticisms that have been raised about the paucity of
work that examines the etiology of self-control and the extent to which self-control theory-
incorporates biological mechanisms or explanations:
…the paucity of research examining the factors that give rise to low self-control is somewhat surprising. We have, for instance, been able to locate only a handful of empirical studies that test them. The findings, in general, have been favorable to Gottfredson and Hirschi’s position that effective child-rearing practices are predictive of self-control in children (but see Cochran, Wood, Sellers, Wilkerson, and Chamlin, 1998) (Wright and Beaver, 2005: 1170).
Theoretically, Gottfredson and Hirschi identify primary socialization effects in the home during
the first decade of life, by parents or caregivers, as key in the development of self-control,
although they also propose secondary socialization agents, such as teachers at schools or schools
themselves. Findings on the importance of parenting processes have not been extensive, often
based on cross-sectional work, but have provided support (Feldman & Weinberger, 1994; Gibbs,
Giever, & Higgins, 1998; Hay, 2001; Polakowski, 1994; Vazsonyi & Huang, 2010). This also
includes work done to test the salience of “secondary” socialization contexts, such as the school
(Turner, Piquero, & Pratt, 2005). However, recently some scholars have called into question how
much do parents really matter in this process (e.g., Beaver, Wright, & Delisi, 2007; Wright &
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Beaver, 2005), with a twin criticism about self-control theory and the extent to which it is valid.
In fact, Wright and Beaver (2005: 1190) conclude:
Gottfredson and Hirschi openly exclude the possibility that self-control has a genetic base. Our study, along with others from various fields, suggests that for self-control theory to be a valid theory of crime it must incorporate a more sophisticated understanding of the origins of self-control (Pratt, Turner and Piquero, 2004; see also Pratt et al., 2002).
Similarly, citing the same work, Cullen, Unnever, Wright, and Beaver note (forthcoming: 17):
Still, Wright and Beaver’s research challenges the parental management thesis in suggesting that the major parental sources of self-control are likely genetic and thus cannot be traced to the style of child-rearing techniques used in a household. There appear to be at least two misunderstandings, related to theoretical interpretation as
well as to the extent to which Wright and Beaver’s important work did in fact show no non-
heritable effects. Together with the previous comment, Gottfredson and Hirschi (1990: 96) in
fact appear to simply deny a denial of biology:
Obviously, we do not suggest that people are born criminals, inherit a gene for criminality, or anything of the sort. In fact, we explicitly deny such notions (see Chapter 3). What we do suggest is that individual differences may have an impact on the prospects of effective socialization (or adequate control). Effective socialization is, however, always possible whatever the configuration of individual.
Individual differences imply biological or genetic variability which they describe as primary and
as existing prior to any socialization processes or effects children might experience:
“Two general sources of variation are immediately apparent in this scheme. The first is the variation among children in the degree to which they manifest such traits to begin with” and “Obviously, therefore, even at this threshold level the sources of low self-control are complex” (Gottfredson and Hirschi, 1990: 96)
Importantly, and perhaps consistent with theory, Wright and Beaver show in their work and call
attention to the fact that between 55% to 66% of the variability in self-control is heritable, based
on HLM analysis that takes genetic relatedness into account and based on a genetically informed
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estimate of heritability based on the same data. Similarly, Beaver, Connolly, Schwartz, Al-
Ghamdi, and Kobeisy (2013) also found evidence which suggests that much of the observed
stability in self-control over time as well as changes in self-control are related to genetic factors
(78% to 89% and 74% to 92%, respectively). It seems as we have argued previously that
Gottfredson and Hirschi chose to focus on positive socialization effects because these represent
perhaps the only fruitful avenue for meaningful change (perhaps the essence of applied empirical
work in criminology), and research has largely supported this.
In a meta-analysis of 34 rigorous studies focused on testing the malleability of self-
control during the first decade of children’s lives, for instance, Piquero, Jennings and Farrington
(2009) concluded that (1) studies were successful in improving self-control, (2) that these
interventions reduced deviance, and (3) positive effects were robust across a number of potential
moderators, including data sources or raters, clinical versus non-clinical approaches and so forth.
Some larger intervention effects were found for girls in comparison to boys, which the authors
interpreted as being theoretically consistent because girls are less delinquent and because they
are also more receptive to intervention efforts in comparison to boys. Calling for a revised
criminal justice policy, based on their findings about the importance of self-control and its
malleability, they conclude:
Our findings show that interventions aimed at improving socialization and childrearing practices in the first decade of life (e.g., Piquero, Farrington, Welsh, Tremblay, & Jennings, 2009) offer benefits for improving self-control as well as reducing delinquency/crime. It appears that investment in these efforts—in lieu of the more cost-prohibitive incarceration policies of the recent past—should be an important part of the policy response, especially because self-control is malleable and responsive to external sources of socialization (Piquero, Jennings, and Farrington 2009: 28).
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Is Biology Really More Important?
Based on the pioneering work by Wright and Beaver (2005), Beaver, Wright, and Delisi
(2007: 1346) similarly concluded as a basis for their study which focused on the extent to which
neuropsychological motor skill function measures co-varied with self-control that:
“Gottfredson and Hirschi (1990) have dismissed the possibility that self-control has a biological or genetic component (Wright and Beaver, 2005) and to take these findings into consideration, the explanation of self-control set forth by Gottfredson and Hirschi needs to be revised with an explicit focus on how the biological processes of the brain affect the development of self-control”.
The study provided evidence that measures of fine and gross motor skills were negatively
associated with measures of self-control, as were socialization variables, based on a random
sample of 3,000 young children. This was found for both boys and girls part of the ECLS-K,
although parenting measures did not reach significance for girls when prior self-control was
added to model tests in the prediction of 1st grade self-control (only a test of developmental
changes in self-control from kindergarten to 1st grade). There is little doubt that the frontal cortex
is very important in the process of decision making (e.g., Renya, Chapman, Dougherty, &
Confrey, 2011); we know from recent brain imaging work that these decision centers continue to
mature into the early 20s and that it would be important and interesting to integrate these insights
into our understanding of how to explain crime and deviance, particularly among youth, and
perhaps to elaborate on self-control theory.
However, the extent to which neurological deficits per se are generally important for
crime and deviance, something Moffitt and colleagues also focused on in the 1990s, or to what
extent brain maps or mapping provides novel insights into why people deviate from social
norms, is less clear. Mapping the brain and understanding that it continues to develop past
adolescence, understanding that it is involved and which regions of it are involved in decision
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making are important. But, what are we are looking for exactly? This search at times is
reminiscent of when biology and biological explanations were considered to be more central in
our understanding of crime and deviance; yet, whether at the molecular, genetic level or
physiological processes observed in the brain, having an idea of what we are looking for would
seem to be a hallmark of what is necessary to building knowledge, understanding, or a coherent
and testable theory of crime and deviance. Consider the following comment by expert
neuroscientists who study neurological control processes:
In sum, control functions that are typically associated with control (e.g., inhibition, task setting, and updating) are defined largely based on behavioral observations (Miyake, Friedman, Emerson, Witzki, & Howerter, 2000). How these functions correspond to distinct indicators of brain activity will determine whether they are truly distinct componential entities or whether they emerge from the interactions of various systems in the brain and are therefore manifest only in the minds of cognitive scientists (Lenartowicz, Kalar, Congdon, & Poldrack, 2010: 690). One of the studies cited by Beaver, Wright, and Delisi (2007) as the basis and impetus for
their work was research done by Cauffman, Steinberg, and Piquero (2005), which juxtaposed
self-control measures with physiological measures related to autonomic nervous system
functioning (heart rate) as well as measures of intelligence, and neuropsychological functioning
(spatial span, a measure of cerebral lateralization). In a comparison of incarcerated youth (2/3 of
whom had committed a violent crime) and a sample of high school students from the general
population, they found that only a measure of self-control and parental education predicted
antisocial behaviors in the high school sample. On the other hand, a measure of self-control,
heart rate, and the measure of brain lateralization predicted antisocial behaviors among
incarcerated youth. Cauffman, Steinberg, and Piquero (2005: 155) conclude:
“In sum, while one component of self-control, future orientation, remains significant in this full model, it does not render insignificant the effects of neuropsychological functioning (in the form of spatial span scores) and biological
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factors (in the form of heart rate). Thus, while certain components of self-control are important in distinguishing the two groups, it does not eliminate other factors that Gottfredson and Hirschi would expect to be eliminated. These results, then, support the contentions of those scholars attributing importance to neuropsychological and biological factors in addition to self-control (Moffitt, 1993; Rowe, 2002). There is no question that this study provided an interesting insight into potential
biologically-linked processes in the explanation of variability in adolescent deviance and crime.
At the same time, it is simply unclear the extent to which the findings provide new and coherent
information that stands in effect to invalidate self-control theory as some have suggested. It is
also not clear whether the theory has any predictions related to heart rate or brain lateralization as
found in this latter study; these were not found to be important for antisocial behaviors among
adolescents from the general population. It is perhaps self-evident that Gottfredson and Hirschi
did not discuss issues related to heart rate or brain lateralization; but does this provide the basis
for a revised or different theoretical explanation?
There is no doubt that the group of incarcerated youth, where 2/3 were in jail because
they killed, assaulted or raped another human being, might suffer from neurological and other
deficits. These youth seem particularly troubled, perhaps suffering from organic deficits. Yet,
self-control was important in understanding their antisocial behaviors as theorized. This means
they suffered from self-control deficits as well as other intellectual, emotional, and perhaps
neurological ones. Gottfredson and Hirschi consider low self-control a probabilistic individual
characteristic important in understanding variability in crime and deviance. They do not seem to
eliminate biology as pointed out, nor do they address or exclude serious intellectual impairments,
perhaps organic in origin or any other individual characteristics. They note on the probabilistic
nature of self-control:
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In our view, lack of self-control does not require crime and can be counteracted by situational conditions or other properties of the individual. At the same time, we suggest that high self-control effectively reduces the possibility of crime – that is, those possessing it will be substantially less likely at all periods of life to engage in criminal acts (Gottfredson and Hirschi: 89).
They also specifically comment on other known correlates and predictors of crime, including
“low intelligence, high activity level, physical strength, and adventuresomeness” (Gottfredson
and Hirschi, 1990: 96) and how these are known to interfere with or support effective
socialization of self-control, with the development of self-control.
Both Biology and Socialization are Important!
Beaver et al. (2013) showed that mostly genetic and non-genetic (shared and non-shared)
effects are important in understanding the development of self-control and its stability over time.
Similarly, Beaver, Ratchford, and Ferguson (2009) illustrate that although a molecular marker
had no direct effect on self-control, the interaction between the 5HTTLPR allele and time spent
with delinquent synergistically explained variability in levels of self-control. Delisi (2011)
recently wrote a poignant editorial about the importance of biology given many of these
reviewed insights, calling for more genetically or biologically informed scholarship focused on
the etiology of crime and deviance. He notes:
For decades, criminology viewed genetics as startling, unconventional, and embarrassing. Genetics was l'enfant terrible. Today, a criminology that ignores or disputes the salience of genes toward understanding, preventing, and treating antisocial behavior risks being embarrassed (DeLisi, 2013: 515).
True, l’enfant terrible, for many reasons; but this is also simply related to recent advances in
molecular biology, in neuroimaging, or in the application of advanced quantitative techniques
that are based on very narrowly defined and not readily obtained twin and adoption studies (e.g.,
the Add Health which was planned over 20 years ago). But is it really so bad? Given the
explanation in the present chapter about the primacy and full acknowledgement of individual
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differences that are present at birth, according to Gottfredson and Hirschi, this might simply be a
case of misattributing a purely environmental position to their work, to their theoretical work.
Few would argue today or even twenty years ago, in the 1990s, that socialization is in
fact a one-way street; rather, we have known it is a two-way street, due to individual,
biologically-based differences; it is a dynamic and complex process, much as detailed by Sandra
Scarr in her 1992 SRCD presidential address, about genotype -> environment effects, about
behavior genetic evidence, about “good enough parenting,” or about the salience of non-shared
environmental effects on child and adolescent development. These are, nevertheless,
environmental effects, or as Plomin and Thomson (1987) describe them, “micro-environments
within families.” It is certainly true that criminologists have eschewed even speaking of biology
or biologically informed explanations, and yet, they were there, even in the General Theory of
Crime, including how Gottfredson and Hirschi approach the importance or un-importance of
culture, of cross-cultural, comparative criminology (Chapter 8). They argue that adopting
environmental, at time idiosyncratic, explanations of crime, driven by positivist thinking has led
to a fragmented field that literally did not know what it was looking for. Rather, they call for a
culture-free, universalist conceptualization of crime and delinquency, one that makes a number
of implicit assumptions about human nature and human behavior, more consistent with a
classical conception of crime, and one that transcends local mores, practices and environments
(Vazsonyi et al., 2011; 2004).
In a comprehensive review of behavior genetic work on antisocial behavior, Moffitt
(2005) concludes that both genetic and environmental causes are equally important for antisocial
outcomes. Much of her early developmental work on the etiology of antisocial or deviant
behaviors focused on neurological deficits as well as a typology of adolescence-limited versus
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lifecourse persistent offenders, related to her interpretation of the age-crime distribution of crime.
Consider the following explanation:
Gottfredson and Hirschi’s (1990) general theory suggests that a single intervention strategy aimed at increasing self-control would suffice to prevent antisocial behavior at any and all ages. In contrast, Moffitt’s (1993) developmental theory implies that one set of intervention strategies is needed to prevent persistent antisocial behavior that begins in childhood, while another set of strategies is needed to prevent delinquency that emerges in adolescence. Strategies for preventing childhood-onset antisocial behavior would focus on the neurodevelopmental impairments that inhibit the development of verbal abilities and self-control, while strategies preventing adolescent-onset behavior would target the social influences of delinquent peers on behavior (Bartusch, Lynam, Moffit, and Silva, 1997: 42-43). However, more recent work appears to have focused exclusively on the importance of
self-control, with a consideration of both the 1972 birth cohort project in Dunedin New Zealand
as well as the E-risk same-sex twin cohort in the United Kingdom, a behavior genetic study
started in 1998. Perhaps one of the most profound and far reaching recent statements written in
the social scientific literature, Moffitt, Poulton, and Caspi (2013: 359) conclude based on data
from both the Dunedin and the E-Risk behavior genetic project that:
The two cohorts we are studying—born in different countries and different decades—strongly support that improving individual self-control will prove essential for humanity's long-term health, wealth, safety, and happiness [emphasis added].
They also call to action for policies and practices that target the development of self-control
among children by the age of 12:
Improving childhood self-control is especially appealing because we find that differences in self-control between children predict their adult outcomes approximately as well as (sometimes better than) low intelligence and low social class origins, factors known to be extremely difficult to improve through intervention (Moffitt et al., 2013: 57). In the final part of the chapter, we provide a simple illustration of the extent to which
there appears to be an interactive and synergistic process that characterized how parenting, self-
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control, and deviance are related over time in a sample of young children followed from age 4.5
to age 10.5 years.
The Current Study
Vazsonyi and Huang (2010) used the National Institute of Child Health and Human
Development longitudinal study to examine both self-control and deviance developmental
trajectories from ages 4.5 to 10.5. They used simple structural equation modeling (SEM) and
latent growth modeling (LGM) of developmental trajectories, findings suggested that great
construct stability of self-control and deviance over the 6 year period. They found evidence that
children’s self-control increased over the 6 year period, and at the same time, their deviance
trajectories decreased over the same period. However, the longitudinal interrelationships among
parenting, self-control and deviance were not examined in their study. To date, few previous
studies examined longitudinal bidirectional relationships for parenting, self-control and deviance.
We examined bidirectional relationships between parenting and self-control. By
examining the bidirectional or reciprocal relationships between parenting and self-control at ages
4.5, 8.5 and 10.5, we were able to assess the degree to which differences in parenting affect
differences in development of self-control at different age (see Figure 1). In addition to these
bidirectional effects, we also tested how self-control at different ages affected children’s deviant
behaviors. Finally, we were also interested in sex and ethnic/race differences of bidirectional
relationships among parenting, self-control, and deviance.
Methods
Participants
The data for this study were based on the National Institute of Child Health and Human
Development (NICHD) Early Child Care Research Network Study of Early Child Care. Data
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collection commenced in 1991 when participating children were one month old and has
continued until middle adolescence; we focus on assessment completed at ages 4.5, 8.5, and 10.5
years. Over this period, research assistants from the 10 data collection sites have seen each child
at home, in child care, in school, and in a laboratory setting. Families lived in Little Rock, AR;
Irvine, CA; Lawrence, KS, Boston, MA; Philadelphia, PA; Pittsburgh, PA; Charlottesville, VA;
Morganton, NC; Seattle, WA; and Madison, WI. Of the n = 1,364 families that completed initial
data collections at 1 month, complete parent and child data are available on n = 985 families -
when children were in first grade. In comparing the n = 985 families to the n = 379 families
where complete data were unavailable, participant’s mothers were slightly older on average (28.6
years versus 26.38 years), better educated (14.4 years versus 13.7 years) and less likely to be of
minority status (17% versus 27%). The final sample included n= 478 boys and n = 479 girls
(51.1%); most children were European Americans (n= 844 [82.3%]; African American n = 114
[11.1%]; Hispanic N = 22 [1.6%]; Native American n = 40 [4.7%]; Asian n=5 [0.3%]). Thus, we
coded European American as majority group, African American, Hispanic, Native American,
and Asian as children part of the minority group.
Procedures
Information about parents and children was obtained from mothers using face-to-face
interviews and observations of mother child-interactions. Telephone interviews were conducted
numerous times, and classroom data were collected from teachers during the spring semester of
first grade through sixth grade. The entire data collection protocol was reviewed by a steering
committee supervised by the NICHD and was reviewed annually by institutional review boards
of the ten participating institutions responsible for data collection. We focused on three time
points, namely when children were 4.5 years, 8.5 years and 10.5 years old. This was largely a
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function of measurement issues at these time points; measurement of the key study constructs
was consistent, an important prerequisite for longitudinal model tests.
Measure
Parenting. In the current study, though a number of different parenting instruments and
measures are available, the Parent-Child Relationship Scale (Pianta, 1994) was selected, largely
as a function of when they were employed (assessment times) and whether they were repeated
subsequently, to facilitate longitudinal data analysis.
At 4.5 years old, mothers were asked to complete a 30-item questionnaire designed to
assess the target child’s attachment to the parent. The form was adapted from the Student-
Teacher Relationship Scale (Pianta, 2001). Items were rated using a 5 point Likert scale. For
children at 8.5 years old and 10.5 years old parenting was measured by the 30 item Parent-Child
Relationship Scale (Pianta, 1994). Initially these items were selected to measure three
dimensions of warmth/security, anger/dependency, and anxiety/insecurity. Since 3rd and 5th
grade assessments only used 15-items short forms, a 15-item version was employed for the
current study to maintain consistency in measurement over time. The 15-items included the
parent’s feelings and beliefs about his/her relationship with the child as well as items about the
child’s behavior toward the parent. Based on conceptual reasons and an interest in the
warmth/security dimension as well as some data reduction analyses described subsequently, only
eight items were used, each rated by mothers on a 5-point Likert scale ranging from 1= definitely
does not apply to 5 = definitely applies (e.g., “children spontaneously shares personal
information with me”). The reliability estimates for the total Parent-Child Relationship scale
ranged from α = .81 to α =.87 in previous work (e.g., Bradley & Corwyn, 2005). The alpha level
ranged from α =.63 to α=.73 for the current study across the three time points.
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Self-Control. Self-control was measured by the Social Skills Rating System (SSRS,
Gresham & Elliott, 1990). Mothers completed the measure. The SSRS-Parent Form consists of
three parts, namely social skills, problem behaviors, and academic competence scales. The social
skill component includes three subscales, namely cooperation, assertion and self-control. The
current study only focused on the self-control subscale. It includes 10 items rated by mothers on
a 3-point Likert-type scale (0 = never, 1 = sometimes, 2 = very often; e.g., “controls temper
when arguing with other child”). The alpha ranged from α = .82 to α = .87 for the self-control
subscale in previous research (e.g., Bradley & Corwyn, 2005). In the current study, reliabilities
ranged from α = .79 to α = .82 over the three time points.
Deviance. The Child Behavior Checklist (CBCL, parent report) (Achenbach, 1991) was
used to assess children and early adolescent deviant behaviors. The measure includes 33 items
that describe a variety of deviant behaviors, including “lying or cheating,” “steals at home,”
“physically attacks people,” and “gets into many fights”. Mothers rated each item on a 3-point
scale (0 = “not true”, 1 = “somewhat or sometimes true”; and 2 = “very true or often true”). Test-
retest reliability estimates indicated good internal consistency in previous work (statistic α =.76
to α =.93 (Achenbach, 1991). In the current study, reliability estimates were adequate over the
three time points (α =.78 to α =.80).
Results
First, descriptive statistics for the measures were computed. These statistics are presented
in Table 1. They include means, standard deviations, skewness, kurtosis and reliabilities for each
of the measures.
Second, bivariate correlations among parenting, self-control, and deviance were
computed. Listwise deletion was employed for this analysis. Results showed that all the
19
correlations for the three time points were statistically significant and in the expected direction.
Maternal parenting was positively associated with self-control and negatively associated with
deviance, while self-control was negatively associated with deviance (see Table 2).
Third, we specified and tested a longitudinal cross-lagged path model of the relationships
among parenting, self-control, and deviance. Model fit was adequate: χ² = 186.350, df = 16, χ²/df
= 11.647, CFI = 0.951, and RMSEA = 0.088 (90% CI 0.077-0.1) (see Figure 1) more
importantly, results also indicated that parenting at 4.5 years predicted children’s self-control at
8.5 years (β = .136, SE = .024, p <.001), while parenting at 8.5 years did not predict children’s
self-control at 10.5 years (β = .016, SE = .023, p =.482). Interestingly, we also found that
children’s self-control at 4.5 years predicted maternal parenting at 8.5 years (β = .168, SE = .033,
p <.001) and that children’s self-control at 8.5 years predicted maternal parenting at 10.5 years (β
= .065, SE = .031, p <.05), thus providing evidence of a reciprocal process. Findings also
showed that children’s self-control at age 4.5 years negatively predicted children’s deviant
behaviors at 8.5 years (β = -.131, SE = .032, p <.001) and that children’s self-control at 8.5 years
negatively predicted children’s deviant behaviors at age 10.5 years (β = -.116, SE = .026, p
<.001). Lastly, the model also provided evidence that children’s deviant behaviors at 4.5 years
predicted children’s self-control at 10.5 years (β = -.176, SE = .026, p <.001) and that children’s
deviant behaviors at 8.5 years negatively predicted children’s self-control at 10.5 years (β = -
.167, SE .027,p<.001).
Next, we also tested a series longitudinal cross-lagged path models by sex and
ethnicity/race. Results showed that all the model fit are adequately. For male model, χ² =
116,056, df = 16, χ²/df = 7.254, CFI = 0.920, and RMSEA = 0.088 (90% CI 0.095-0.135). For
female model, χ² = 71.38, df = 16, χ²/df = 4.461, CFI = 0.951, and RMSEA = 0.085 (90% CI
20
0.066-0.11). For majority model children the following model fit was observed: χ² = 98.52, df =
16, χ²/df = 6.157, CFI = 0.962, and RMSEA = 0.078 (90% CI 0.064-0.093). On the other hand,
for minority children, the following was found: χ² = 67.798, df = 16, χ²/df = 4.237, CFI = 0.895,
and RMSEA = 0.134(90% CI 0.102-0.168). Results included in Table 3 decompose the effects
by sex and by ethnic/racial group. Findings provide evidence that reciprocal processes were
found between parenting and self-control in both boys and girls during early and middle
childhood (from 4.5 years old to 8.5 years old), but less so during late childhood (8.5 years old to
10.5 years old). Similarly, reciprocal processes were also found between self-control and
deviance in both boys and girls. Finally, in comparisons by ethnic/racial group, findings
indicated that reciprocal processes existed between parenting and self-control among both
majority and minority group children during early and middle childhood (from 4.5 years old to
8.5 years old), but less so during late childhood. However, deviance at age 4.5 years did not
significantly predict children’s self-control four years later at the age of 8.5 years. We found that
an individual’s level of self-control only showed significant effects on parenting during early
childhood (between 4.5 and 8.5 years). At the same time, self-control predicted both parenting
and deviance, while deviance also predicted self-control over time. In general, stability over time
of each of the three constructs was greater in magnitude during late childhood, while at the same
time, the strength of association across them diminished.
Conclusion
In conclusion, self-control remains very important and appears to develop through an
interplay between individual differences and, at least, non-heritable effects. Self-control has
recently been highlighted by some as essential for the well-being and very functioning of
humanity. Much of this work also supports the importance of socialization effects, above and
21
beyond individual differences, thus reaffirming the original theoretical conceptualization of it.
We are optimistic about the theory and its empirical status, the original potent concept of
probabilistic self-control, tempered by situational and contextual opportunities, is important in
our understanding of variability in crime and deviance. Maybe Gottfredson and Hirschi had it
right, after all, when they focused on nurture (without the exclusion of nature) in the
development of self-control. Others tend to agree –scholars conducting sophisticated and state of
the art etiological work over decades and across different continents, with and without the use of
genetically informed designs (Moffitt et al., 2013) as well as scholars extensively synthesizing
rigorous programmatic work addressing the reduction of crime and deviance by addressing self-
control (Piquero et al., 2009). Today, few would argue with the fact that socialization processes
are bidirectional and that they are related to both individual (genetic) differences as much as to
contextual influences. Much like DeLisi (2013) notes, work ignoring this status quo stands a high
chance of embarrassment. However, and thankfully so, today, so far, we can do little about
biological variability, but much about socialization influences and early experiences.
22
Table 1. Descriptive statistics for the main constructs variables
Variable
# items
N Mean SD Skew SE Kurtosis SE α
Parenting at 4.5 years 8 1,077 4.66 0.35 -1.57 0.08 3.38 0.15 0.63
Parenting at 8.5 years 8 1,028 4.25 0.32 -1.32 0.08 4.64 0.15 0.66
Parenting at 10.5 years 8 1,020 4.20 0.35 -1.39 0.07 3.41 0.15 0.73
Self-Control at 4.5 years 10 1,057 1.30 0.31 0.09 0.07 -0.33 0.15 0.79
Self-control at 8.5 years 10 1,028 1.36 0.34 -0.15 0.07 -0.63 0.15 0.82
Self-Control at 10.5 years 10 1,021 1.39 0.33 -0.12 0.07 -0.27 0.15 0.81
Deviance at 4.5 years 8 1,057 0.50 0.35 0.68 0.07 0.26 0.15 0.78
Deviance at 8.5 years 8 1,026 0.37 0.32 0.88 0.08 0.29 0.15 0.79
Deviance at 10.5 years 8 1,020 0.31 0.32 1.20 0.07 1.28 0.15 0.80
23
Table 2. Correlations of Parenting, Self-control, and Deviance
Note. ** Correlations are statistically significant at p < 0.01 (2 tailed)
Variables 1 2 3 4 5 6 7 8 9
1. Parenting (4.5 years)
2. Parenting (8.5 years) .34**
3. Parenting (10.5 years) .29** .49**
4. Self-control (4.5 years) .32** .26** .21**
5. Self-control (8.5 years) .30** .39** .25** .53**
6. Self-control (10.5 years) .23** .29** .28** .49** .70**
7. Deviance (4.5 years) -.19** -.16** -.11** -.39** -.38** -.38**
8. Deviance (8.5 years) -.15** -.22** -.12** -.57** -.51** -.51** .54**
9. Deviance (10.5 years) -.14** -.19** -.24** -.48** -.59** -.59** .51** .70**
24
Table 3: Parameter Estimates for Male versus Female children and for Ethnic/racial Majority versus Minority Children
Male N = 478
Female N = 479
Majority N = 844
Minority N = 181
β SE β SE β SE β SE
Parenting 4.5 years Parenting 8.5 years .242*** .045 .315*** .048 .284*** .035 .258** .091
Parenting 8.5 years Parenting 10.5 years .526*** .052 .497*** .056 .444*** .041 .467*** .084
Self-control 4.5 years Self-control 8.5 years .385*** .058 .452*** .051 .391*** .041 .412*** .095
Self-control 8.5 years Self-control 10.5 years .546*** .045 .593*** .049 .605*** .035 .623*** .073
Deviance 4.5 years Deviance 8.5 years .447*** .057 .484*** .045 .531*** .033 .443*** .075
Deviance 8.5 years Deviance 10.5 years .694*** .040 .602*** .045 .654*** .034 .700*** .075
Parenting 4.5 years Self-control 8.5 years .125** .038 .182*** .039 .170*** .030 .168* .067
Parenting 8.5 years Self-control 10.5 years -.004 .038 .067** .043 .036 .030 -.086 .056
Self-control 4.5 years Parenting 8.5 years .117** .058 .150** .055 .178*** .040 .178** .111
Self-control 8.5 years Parenting 10.5 years .031 .049 .024 .056 .090* .038 .033 .093
Self-control 4.5 years Deviance 8.5 years -.160*** .057 -.151** .052 -.104** .038 -.146** .092
Self-control 8.5 years Deviance 10.5 years -.090* .040 -.140* .046 -.110** .033 -.103* .061
Deviance 4.5 years Self-control 8.5 years -.199*** .045 -.149** .043 -.231*** .035 -.092 .054
Deviance 8.5 years Self-control 10.5 years -.253*** .040 -.120** .046 -.150*** .036 -.241*** .061
25
Figure 1: Path Analysis Parameter Estimates
.466*** .291***
.634*** .484***
.594*** .416***
.168***
.136***
‐.131***
‐.176***
.065*
.016
‐.116***
‐.167***
Parenting 4.5 years
Parenting 8.5 years
Parenting 10.5 years
Self‐Control 4.5 years
Self‐Control 8.5 years
Deviance 8.5 years
Deviance 4.5 years
Self‐Control 10.5 years
Deviance 10.5 years
26
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