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PERUBAHAN SISTEMIKPATOFISIOLOGIGITAtrofi mukosa : apoptosis, vesikulasi mikrovili, kerusakan filamen aktin jaringan OK p< aliran darah di sirkulasi splangnikusPerubahan absorpsi : uptake glukosa & asam amino kerja hepar (kekacauan metabolisme disfungsi >> gagal hati Stress ulcer/ Curlings ulcer OK stress berat + hiperasiditas lambungJANTUNGHipoksia & hipoksemia iskemik miokardiumIskemia GIT stimulasi keluarnya mediator inflamasi (sitokin, TNF-), radikal bebas, & fk depresan miokardium (MDF) OK neutrophile recruitment > mpburuk kerja jantungSISTEM MUSKULARISHipoksia pmecahan glikoprotein massa otot aktivasi siklus urea NO (vasodilator) > kerusakan jaringan (sistem muskularis) atau kondisi spsis

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