Tugas Lc Ulkus Gaster
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Transcript of Tugas Lc Ulkus Gaster
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GASTRIC ULCER
1. Maghrifah Akriani Fifti 12310274
2. Nafla Rintisa 12310317
3.Ni Luh Esti Listiani 12310319
4. Ni Nengah Pini Sriani 12310321
5. Nurhidayati 12310342
6. Yohana Septa Putri 12310493
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once or twice a day because of arthritis in his
knees.
not smoke cigarettes or drink alcohol
pale appearing
he is afebrile,blood pressure is 120/80 mm
Hg,pulse is 95 beats/min, and respiratory rate is14 breaths/min.
Head, eyes, ears, nose, and throat (HEENT)
examination is notable only for pale
conjunctiva.
tenderness in the epigastrium.
dark black stool that is strongly fecal occult
blood test (FOBT) positive.
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Definition
Gastric ulceris an open wound in the
side of edema with induration ulcer base
covered with debris.
Gastric ulcers are sores in the lining ofthe stomach . Gastric ulcers can be treated
.A small portion of these ulcers may
become cancerous.
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Epidemiology
In the United States , peptic ulcer disease
( PUD ) affects about 4.5 million people
each year with 20 % caused by
Helicobacter pylori . Prevalence of gastriculcers in men is 11-14 % , and the
prevalence in women is 8-11%.
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Etiology
According to Sam LK ( 1994 ) there areseveral factors etiology of gastric ulcers
that Helicobacter pylori infection , NSAID
use , smoking, and habits food .
1. Helicobacter pylori
It has long been known to have arelationship with gastric ulcer infection
germs Helicobacter pylori
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2. NSAID
The use of these drugs interfere with infiltration
of the mucosa, destroying the mucosal and cause
mucosal damage 30% of adults who use NSAIDs
have adverse GI .
3. Genetic
More than 20 % of patients have a family history
of gastric ulcers.
4. Additional etiologic factors
In addition , one rather than the disease may be
related to gastric ulcersie liver cirrhosis , chronic
obstructive pulmonary disease and autoimmune
diseases.
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Symptoms
Symptoms depend on the location and age of
ulcer patients , especially patientselderly
often have little or no symptoms . Pain is the
most common often in the form of epigastricpain and reduced in the presence of food
oradministration of antacids . The pain may
be burning, or sometimes as the sensation of
hunger . The pain is usually chronic andrecurrent . only abouthalf of the patients
present with symptoms typical .
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Classification
By Region/Location :
1. Stomach
2. Duodenum
3. Esophagus
4. MeckelsDiverticulum
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Pathogenesis
Duodenal ulcers are different with gastric ulcers: Duodenal ulcersand gastric acid plays a role in
the increase of HCl and the stimulation of the
basal state or after a meal.
Gastric ulceris more important mucosal
defense ( defensive factors ) ; include gastric
motility disorders that cause reflux of bile from
the duodenum into the stomach , slowing
gastric emptying
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Treatment
1. Antacids
2. The management of diet, by eating a little bit butwith a high frequency (often), to neutralize
excess acid in peptic ulcer.
3. Anticholinergic, inhibiti the parasympatheticpostganglionic nerves that secrete achetilcholine.
However, this drug is rarely used due to other
effects that are less favorable hull, which would
inhibit the motility of the intestine alone will
result in slowing of gastric emptying alone.
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4. Inhibiting H2 (Histamine) because effects
of histamine stimulates acid secretion /HCl and pepsinogen. Preparations used
were cimetidine, ranitidine, and famotidin
5. Antimicroba / anti-bacterial, for example,if the presence of Helicobacter pylori
infection.
6. Get plenty of rest, both physically andemotionally
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